Reference : Guillain-Barré syndrome following hepatitis E
Scientific journals : Article
Human health sciences : Gastroenterology & hepatology
http://hdl.handle.net/2268/66820
Guillain-Barré syndrome following hepatitis E
English
Loly, Jean-Philippe [Université de Liège - ULg > > Frais communs médecine >]
Rikir, Estelle mailto [Université de Liège - ULg > > Neurologie Sart Tilman >]
Seivert, Maxime [Université de Liège - ULg > > Frais communs médecine >]
Legros, E. [Verviers > > General Practitioner > >]
Defrance, P. [CHPLT La Tourelle > Hepato-Gastroenterology > > >]
Belaiche, Jacques mailto [Université de Liège - ULg > > Gastro-Entérologie-Hépatologie >]
Moonen, Gustave mailto [Université de Liège - ULg > > Neurologie Sart Tilman >]
Delwaide, Jean mailto [Université de Liège - ULg > > Gastro-Entérologie-Hépatologie >]
2009
World Journal of Gastroenterology
W J G Press
15
13
1645-1647
Yes (verified by ORBi)
International
1007-9327
Beijing
China
[en] Gangliosides ; Guillain-Barré syndrome ; Hepatitis E ; Molecular mimicry ; Viral hepatitis
[en] Guillain-Barré syndrome (GBS) is often triggered by a
preceding bacterial or viral infection. Occasionally, it
has been observed in association with acute hepatitis
A, B and C, and three cases have been previously
described in India in which GBS was associated with
acute hepatitis E. A molecular mimicry mechanism
is supposed to be involved in the pathogenesis
of GBS triggered by infectious agents, although
the nature of the shared epitopes has not been
characterized in most instances, including that in the
case of hepatotropic viruses. We report a case of GBS
following acute hepatitis E in a European individual.
The presence of antiganglioside GM2 antibodies in
this patient suggested molecular mimicry involving
ganglioside GM2 in the pathogenesis of GBS associated
with hepatitis E.
Researchers ; Professionals ; Students ; General public
http://hdl.handle.net/2268/66820
10.3748/wjg.15.1645

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