[en] Previous studies have described the presence of glycine receptor mRNA during early stages of
embryonic cortex development. Here, we have tested the functionality of those receptors in
migratory interneurons and demonstrated their involvement in the control of cell migration. We
suggest a mechanism whereby activation of glycine receptors during tangential migration
activates voltage gated calcium channels and favors influx of calcium that ultimately affect
myosin II activity, a mechanism that fine tune nuclear translocation and thus migration speed.
