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See detailLorsque la poésie fait le souverain. Etude sur la Chanson de la Croisade albigeoise
Raguin, Marjolaine ULiege

Book published by Honoré Champion (2015)

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See detailLorsque le management entre au tribunal: évolution ou révolution ?
Schoenaers, Frédéric ULiege

in Revue de Droit de l'ULB (2014), 41

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See detailLorsque les limites des sciences et les sentiers de chasse s’entrecroisent: Une ethnographie des laboratoires scientifiques amazoniens en partenariat avec les communautés autochtones locales
Escobar Jimenez, Kelly ULiege

Doctoral thesis (2012)

Currently, protected areas and protected natural species are in a state of proliferation all over the world. Landscapes, flagship species, peoples in voluntary isolation, ecosystems, and “biological ... [more ▼]

Currently, protected areas and protected natural species are in a state of proliferation all over the world. Landscapes, flagship species, peoples in voluntary isolation, ecosystems, and “biological corridors” are all protected. In order to achieve this, categories of protection at different geographical levels have been mobilized. The processes that allow any one of these categories to become operational within a precise territory are more or less well standardized and have in common, amongst others things, two components that have caught my attention: Firstly, the central role of previous scientific and technical studies, and secondly, the desire to ensure the participation of the populations concerned. How are relations between scientists and the non-scientific public in participatory environmental management? This thesis seeks to answer this question, from the starting point of an analysis of the cognitive dimension of participatory environmental management, i.e. how the production of knowledge affects the modes of existence within protected areas (and consequently their human and nonhuman inhabitants). Building upon the study of daily activities that bring scientists and indigenous peoples into contact in and around the scientific field stations “Yasuní” and “Juri Juri Kawsay” (in the Ecuadorian Amazon), this research describes the relationship between ways of knowing a conflicting object (for example a natural species, a territorial boundary, an isolated people) and the consequences in terms of configuration of that object (in terms of identity, value, and fragility) and its management (administration, use, protection, recovery or abandonment). By mobilizing the approach of Actor-Network theory, this dissertation attempts to outline how mediation between humans and nonhumans, non-scientific and scientific, occurs. It also offers a reflection on how the Agency of an entity (the competence to make make distributed in the humans and nonhumans actors) comes to influence the joint actions. This research can capture the ambiguity, instability, resistance and complexity that still exist in the Amazon, with some unexpected findings along the way, by showing the changes among things and people throughout development projects, land management initiatives, and conservation and science programs. After describing the trajectory of four entities (laboratories, species conservation, territories and subjects), which meet at the Amazonian interfaces where science, conservation and development are in competition, the findings of this dissertation have met two ambitions: to consider the importance of taking into account the complexity and historicity of scientific data and to answer a broader question: What are the relevant factors to take into account when considering the possibilities for joint action between scientists and indigenous populations? [less ▲]

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See detailLorsque Wolfgang Amadeus Mozart découvrît les sites archéologiques du Gofle de Naples
Morard, Thomas ULiege

Scientific conference (2015, October 09)

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See detailLos caminos de Santiago. Aller en pèlerinage à Compostelle
George, Philippe ULiege; Stiennon, Jacques ULiege

in Liège. Autour de l'an mil, la naissance d'une principauté (Xe-XIIe siècle) (2000)

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See detailLos editores españoles y la traducción en la Argentina: desembarco en tierras fértiles
Willson, Patricia ULiege

in Pagni, Andrea (Ed.) El exilio republicano español en México y Argentina. Historia cultural, instituciones literarias, medios (2011)

Since 1938, the year in which Losada publishing house was founded in Buenos Aires, publishers of Spanish origin contributed to a diversified and autonomous cultural field where translation played already ... [more ▼]

Since 1938, the year in which Losada publishing house was founded in Buenos Aires, publishers of Spanish origin contributed to a diversified and autonomous cultural field where translation played already a major role. This chapter examines the practices and agents of literary import, the different figures of translator, and the question of so-called «national tongue». [less ▲]

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See detailLos relatos secantes de José Muñoz y Carlos Sampayo / Les Récits sécants de José Muñoz et Carlos Sampayo
Dejasse, Erwin ULiege

in Vazquez, Laura (Ed.) Actas del Segundo Congreso Internacional Viñetas Serias: narrativas gráficas: lenguajes entre el arte y el mercado (2012, September 26)

The comics of Muñoz and Sampayo are full of strange characters who surprise by their enigmatic appearance but do not seem to play a particular role in the overall framework of the story. This essay ... [more ▼]

The comics of Muñoz and Sampayo are full of strange characters who surprise by their enigmatic appearance but do not seem to play a particular role in the overall framework of the story. This essay attempts to determine their nature, how they are articulated within the web formed by all the panels and to analyze how they make sense within the context of the oeuvre in its entirety. [less ▲]

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See detailLos(t)s in Transitions: How Diverse Families Are Grieving and Struggling to Achieve a New Identity
D'Amore, Salvatore ULiege; Scarciotta, Lidia ULiege

in Journal of Family Psychotherapy (2011), 22

Diverse families are constantly confronted with multiple losses. In psychotherapeutic consultation, they often appear as “grieving” systems whose identity is built on a loss. New theories of grief focus ... [more ▼]

Diverse families are constantly confronted with multiple losses. In psychotherapeutic consultation, they often appear as “grieving” systems whose identity is built on a loss. New theories of grief focus on family meaning-making processes. Unresolved grieving entails limited emotion sharing as well as poor family meaning-making. Moreover, some factors inhibit this process while others stimulate it. Using clinical illustration, this article tries to prove the relevance of such concepts. It suggests working to re-create, review, and reconstruct the family ties around its losses or break-ups. However, this work would remain reductive if it would not take account of the benefits and gains that often, within the experience of grief, families do not consider or anticipate. [less ▲]

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See detailLoss in risk-taking: Absence of optimal gain or reduction in one’s own resources?
Anselme, Patrick ULiege

in Behavioural Brain Research (2012), 229(2), 443-446

Determining how living beings react to tasks that reflect realistic situations of risk has given rise to a vast literature. However, I argue that the methodologies traditionally used to test humans and ... [more ▼]

Determining how living beings react to tasks that reflect realistic situations of risk has given rise to a vast literature. However, I argue that the methodologies traditionally used to test humans and nonhumans relative to risk often fail to achieve their goal. When risk is modelled in laboratory, potential decision cost (or potential loss) typically denotes an absence of optimal gain. In contrast, when risk occurs in real-life situations, potential loss denotes the reduction in an individual’s limited resources – whether energetic, social, financial, etc. This conceptual difference about the nature of risk may have important implications for the understanding of the parameters that control risk-taking behaviour. [less ▲]

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See detailLoss of axin2 in murine aortic valves leads to myxomatous disease
Hulin, Alexia ULiege; Alfieri, Christina M.; Yutzey, Katherine E.

Poster (2015)

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See detailLoss of Axin2 leads to Myxomatous Valve Disease
Hulin, Alexia ULiege; Yutzey, Katherine

Poster (2016)

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See detailLoss of Axin2 results in impaired heart valve maturation and subsequent myxomatous valve disease.
Hulin, Alexia ULiege; Moore, Vicky; James, Jeanne M. et al

in Cardiovascular Research (2017), 113(1), 40-51

AIMS: Myxomatous valve disease (MVD) is the most common aetiology of primary mitral regurgitation. Recent studies suggest that defects in heart valve development can lead to heart valve disease in adults ... [more ▼]

AIMS: Myxomatous valve disease (MVD) is the most common aetiology of primary mitral regurgitation. Recent studies suggest that defects in heart valve development can lead to heart valve disease in adults. Wnt/beta-catenin signalling is active during heart valve development and has been reported in human MVD. The consequences of increased Wnt/beta-catenin signalling due to Axin2 deficiency in postnatal valve remodelling and pathogenesis of MVD were determined. METHODS AND RESULTS: To investigate the role of Wnt/beta-catenin signalling, we analysed heart valves from mice deficient in Axin2 (KO), a negative regulator of Wnt/beta-catenin signalling. Axin2 KO mice display enlarged mitral and aortic valves (AoV) after birth with increased Wnt/beta-catenin signalling and cell proliferation, whereas Sox9 expression and collagen deposition are decreased. At 2 months in Axin2 KO mice, the valve extracellular matrix (ECM) is stratified but distal AoV leaflets remain thickened and develop aortic insufficiency. Progressive myxomatous degeneration is apparent at 4 months with extensive ECM remodelling and focal aggrecan-rich areas, along with increased BMP signalling. Infiltration of inflammatory cells is also observed in Axin2 KO AoV prior to ECM remodelling. Overall, these features are consistent with the progression of human MVD. Finally, Axin2 expression is decreased and Wnt/beta-catenin signalling is increased in myxomatous mitral valves in a murine model of Marfan syndrome, supporting the importance of Wnt/beta-catenin signalling in the development of MVD. CONCLUSIONS: Altogether, these data indicate that Axin2 limits Wnt/beta-catenin signalling after birth and allows proper heart valve maturation. Moreover, dysregulation of Wnt/beta-catenin signalling resulting from loss of Axin2 leads to progressive MVD. [less ▲]

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See detailLoss of beta-catenin promotes chondrogenic differentiation of aortic valve interstitial cells.
Fang, Ming; Alfieri, Christina M.; Hulin, Alexia ULiege et al

in Arteriosclerosis, thrombosis, and vascular biology (2014), 34(12), 2601-8

OBJECTIVE: The Wnt/beta-catenin signaling pathway has been implicated in human heart valve disease and is required for early heart valve formation in mouse and zebrafish. However, the specific functions ... [more ▼]

OBJECTIVE: The Wnt/beta-catenin signaling pathway has been implicated in human heart valve disease and is required for early heart valve formation in mouse and zebrafish. However, the specific functions of Wnt/beta-catenin signaling activity in heart valve maturation and maintenance in adults have not been determined previously. APPROACH AND RESULTS: Here, we show that Wnt/beta-catenin signaling inhibits Sox9 nuclear localization and proteoglycan expression in cultured chicken embryo aortic valves. Loss of beta-catenin in vivo in mice, using Periostin(Postn)Cre-mediated tissue-restricted loss of beta-catenin (Ctnnb1) in valvular interstitial cells, leads to the formation of aberrant chondrogenic nodules and induction of chondrogenic gene expression in adult aortic valves. These nodular cells strongly express nuclear Sox9 and Sox9 downstream chondrogenic extracellular matrix genes, including Aggrecan, Col2a1, and Col10a1. Excessive chondrogenic proteoglycan accumulation and disruption of stratified extracellular matrix maintenance in the aortic valve leaflets are characteristics of myxomatous valve disease. Both in vitro and in vivo data demonstrate that the loss of Wnt/beta-catenin signaling leads to increased nuclear expression of Sox9 concomitant with induced expression of chondrogenic extracellular matrix proteins. CONCLUSIONS: beta-Catenin limits Sox9 nuclear localization and inhibits chondrogenic differentiation during valve development and in adult aortic valve homeostasis. [less ▲]

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See detailLoss of calcyphosin gene expression in mouse and other rodents
Clément, S.; Dumont, J.; Schurmans, Stéphane ULiege

in Biochemical and Biophysical Research Communications (1997), 232

alcyphosine-for calcium binding and regulated by cyclic AMP through phosphorylation protein-is a target of both the cyclic AMP and the Ca(+2)-phophatidylinositol cascades first isolated from dog thyroid ... [more ▼]

alcyphosine-for calcium binding and regulated by cyclic AMP through phosphorylation protein-is a target of both the cyclic AMP and the Ca(+2)-phophatidylinositol cascades first isolated from dog thyroid, and then from rabbit and human brain. Although the exact function of this 24kD protein is unknown, calcyphosine could be implicated in the cross-signaling between these cascades to coordinate cellular proliferation and differentiation. Here, we report the sequence of a pseudogene which is the murine calcyphosine homologue, and demonstrate that it represents the unique sequence homologous to the dog calcyphosine gene in the murine genome. The lack of expression of this murine pseudogene in brain and thyroid-two major sites of dog calcyphosine expression-was extended to 5 other rodents, and suggest the existence of alternative pathway(s) to fill the function of calcyphosine in rodents [less ▲]

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See detailThe loss of cell-cell adhesion molecules is associated with an increased vimentin expression in migrating human bronchial cells.
Buisson, AC; Gilles, Christine ULiege; Polette, M et al

Poster (1996, September 08)

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See detailLoss of Elp3 Impairs the Acetylation and Distribution of Connexin-43 in the Developing Cerebral Cortex.
Laguesse, Sophie; Close, Pierre ULiege; Van Hees, Laura ULiege et al

in Frontiers in Cellular Neuroscience (2017), 11

The Elongator complex is required for proper development of the cerebral cortex. Interfering with its activity in vivo delays the migration of postmitotic projection neurons, at least through a defective ... [more ▼]

The Elongator complex is required for proper development of the cerebral cortex. Interfering with its activity in vivo delays the migration of postmitotic projection neurons, at least through a defective alpha-tubulin acetylation. However, this complex is already expressed by cortical progenitors where it may regulate the early steps of migration by targeting additional proteins. Here we report that connexin-43 (Cx43), which is strongly expressed by cortical progenitors and whose depletion impairs projection neuron migration, requires Elongator expression for its proper acetylation. Indeed, we show that Cx43 acetylation is reduced in the cortex of Elp3cKO embryos, as well as in a neuroblastoma cell line depleted of Elp1 expression, suggesting that Cx43 acetylation requires Elongator in different cellular contexts. Moreover, we show that histones deacetylase 6 (HDAC6) is a deacetylase of Cx43. Finally, we report that acetylation of Cx43 regulates its membrane distribution in apical progenitors of the cerebral cortex. [less ▲]

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