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See detail13ième activité du CDS - Evaluation formative
Colaux, Catherine ULg

Report (2007)

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See detail14 steps to writing clearly
Van Damme, Hendrik ULg; Ceelen, W.; Detry, Olivier ULg et al

in Acta Chirurgica Belgica (2009), 109

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See detailA 14,000 yr elemental and lead isotopis record in a peat core in the south of Poland –results and interpretation
Tudyka, K.; De Vleeschouwer, F.; Pazdur, A. et al

Poster (2010)

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See detail14ième activité du CDS -Prérequis
Colaux, Catherine ULg

Report (2008)

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See detail14th Annual meeting of the Belgian Transplantation Society - March 27, 2007, Brussels, Belgium - Preface
Detry, Olivier ULg; Donckier, Vincent

in Transplantation Proceedings (2007), 39(8), 2633-2633

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See detailThe 14th International Conference on Human Retrovirology: HTLV and related retroviruses (July 1-4, 2009; Salvador, Brazil).
Willems, Luc ULg

in Retrovirology (2009), 6

The "14th International Conference on Human Retrovirology: HTLV and Related Retroviruses" was held in Salvador, Bahia, from July 1st to July 4th 2009. The aim of this biennial meeting is to promote ... [more ▼]

The "14th International Conference on Human Retrovirology: HTLV and Related Retroviruses" was held in Salvador, Bahia, from July 1st to July 4th 2009. The aim of this biennial meeting is to promote discussion and share new findings between researchers and clinicians for the benefit of patients infected by human T-lymphotropic virus (HTLV). HTLV infects approximately 15-20 million individuals worldwide and causes a broad spectrum of diseases including neurodegeneration and leukemia. The scientific program included a breadth of HTLV research topics: epidemiology, host immune response, basic mechanisms of protein function, virology, pathogenesis, clinical aspects and treatment. Exciting new findings were presented in these different fields, and the new advances have led to novel clinical trials. Here, highlights from this conference are summarized. [less ▲]

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See detail15 Minutes about SAXS and gels
Gommes, Cédric ULg; Goderis, Bart; Pirard, Jean-Paul ULg

Scientific conference (2009, May)

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See detail15-deoxy-delta12,14-prostaglandin J2 inhibits Bay 11-7085-induced sustained extracellular signal-regulated kinase phosphorylation and apoptosis in human articular chondrocytes and synovial fibroblasts
Relic, Biserka ULg; Benoit, Valerie; Franchimont, Nathalie et al

in Journal of Biological Chemistry (2004), 279(21), 399-403

We have previously shown that nuclear factor-kappaB inhibition by adenovirus expressing mutated IkappaB-alpha or by proteasome inhibitor increases human articular chondrocytes sensibility to apoptosis ... [more ▼]

We have previously shown that nuclear factor-kappaB inhibition by adenovirus expressing mutated IkappaB-alpha or by proteasome inhibitor increases human articular chondrocytes sensibility to apoptosis. Moreover, the nuclear factor-kappaB inhibitor BAY11-7085, a potent anti-inflammatory drug in rat adjuvant arthritis, is itself a proapoptotic agent for chondrocytes. In this work, we show that BAY 11-7085 but not the proteasome inhibitor MG-132 induced a rapid and sustained phosphorylation of extracellular signal-regulated kinases (ERK1/2) in human articular chondrocytes. The level of ERK1/2 phosphorylation correlated with BAY 11-7085 concentration and chondrocyte apoptosis. 15-Deoxy-delta(12,14)-prostaglandin J2 (15d-PGJ2) and its precursor prostaglandin (PG) D2 but not PGE2 and PGF2alpha rescued chondrocytes from BAY 11-7085-induced apoptosis. 15d-PGJ2 markedly inhibited BAY 11-7085-induced phosphorylation of ERK1/2. BAY 11-7085 also induced ERK1/2 phosphorylation and apoptosis in human synovial fibroblasts, and these reactions were down-regulated by 15d-PGJ2. Further analysis in synovial fibroblasts showed that only molecules that suppressed BAY 11-7085-induced phosphorylation of ERK1/2 (i.e. 15d-PGJ2, PGD2, and to a lesser extent, MEK1/2 inhibitor UO126, but not prostaglandins E2 and F2alpha or peroxisome proliferator-activated receptor-gamma agonist ciglitazone) were able protect cells from apoptosis. These results suggested that the antiapoptotic effect of 15d-PGJ2 on chondrocytes and synovial fibroblasts might involve inhibition of ERK1/2 phosphorylation. [less ▲]

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See detail1513. L'année terrible. Le siège de Dijon.
Vissière, Laurent; Marchandisse, Alain ULg; Dumont, Jonathan ULg

Book published by Faton (2013)

A l’été 1513, la France fut menacée par une redoutable coalition européenne. Pendant que les Anglais et les Impériaux attaquaient au nord, une importante armée suisse descendait sur la Bourgogne et venait ... [more ▼]

A l’été 1513, la France fut menacée par une redoutable coalition européenne. Pendant que les Anglais et les Impériaux attaquaient au nord, une importante armée suisse descendait sur la Bourgogne et venait mettre le siège devant la cité de Dijon, défendue par le gouverneur de la province, Louis II de La Trémoille. Après plusieurs jours de bombardement, la situation devint critique et les Suisses étaient prêts à monter à l’assaut des murailles éboulées, lorsque La Trémoille obtint in extremis de négocier avec eux. Contre une forte indemnité de guerre et quelques promesses, les envahisseurs acceptèrent de rentrer chez eux, renonçant à continuer une guerre qui s’annonçait désastreuse non seulement pour Dijon et la Bourgogne, mais pour le royaume tout entier. Le départ, totalement inattendu des Suisses, fut perçu par les Dijonnais comme un véritable miracle, qu’ils attribuèrent à l’intercession de Notre-Dame de Bon-Espoir, une Vierge romane, vénérée en l’église Notre-Dame. C’est en son honneur qu’ils organisèrent une procession annuelle à travers les rues de la cité. C’est aussi en son honneur qu’un échevin, Philibert Godran, commanda une immense tapisserie commémorative, toujours conservée au musée des Beaux-Arts et récemment restaurée. Pour l’anniversaire du 500e anniversaire de la levée du siège de Dijon (septembre 2013), date qui correspond aussi à la réouverture du musée des Beaux-Arts et à la nouvelle présentation de la tapisserie, il nous a paru intéressant de revenir sur cet événement aujourd’hui bien oublié ou jugé anecdotique. Il ne s’agit pas de traiter le seul point de vue dijonnais. L’invasion de la Bourgogne et le siège de Dijon sont à replacer dans leur véritable contexte – celui des guerres d’Italie et des guerres européennes qui ont suivi. Le fil directeur de l’ouvrage est la tapisserie des Suisses, un chef-d’oeuvre qui mesure près de huit mètres de long, et qui relate chaque scène du siège tout en en présentant les principaux protagonistes. La tapisserie offre aussi la plus ancienne représentation connue de la ville de Dijon. L’analyse méthodique de cette oeuvre exceptionnelle, jointe au dépouillement des très riches archives communales de la ville permet d’offrir une vision renouvelée du siège de 1513, mais également de la vie bourguignonne en ce début du XVIe siècle. Les récentes fouilles menées à l’occasion des travaux du tramway fournissent en outre l’occasion de proposer un point sur les fortifications disparues de Dijon et une carte archéologique de la cité au moment du siège. [less ▲]

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See detail15ième activité du CDS - Apprentissage langues
Colaux, Catherine ULg

Report (2008)

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See detail15th Benelux Congress of Zoology abstract book
Colleye, Orphal ULg; Dauby, Patrick ULg; Fabri, Gregory et al

Book published by Editions de l'université de Liège (2008)

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See detail§16 Aristote. Tableau récapitulatif des occurrences. Observations
Gavray, Marc-Antoine ULg

in Motte, André; Somville, Pierre; Gavray, Marc-Antoine (Eds.) et al Ousia dans la philosophie grecque des origines à Aristote. Travaux du Centre d’études aristotéliciennes de l’Université de Liège (2008)

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See detail16-kDa prolactin and bromocriptine in postpartum cardiomyopathy.
Hilfiker-Kleiner, Denise; Struman, Ingrid ULg; Hoch, Melanie et al

in Current Heart Failure Reports (2012), 9(3), 174-82

Peripartum cardiomyopathy (PPCM) is a potentially life-threatening heart disease emerging toward the end of pregnancy or in the first postpartal months in previously healthy women. Recent data suggest a ... [more ▼]

Peripartum cardiomyopathy (PPCM) is a potentially life-threatening heart disease emerging toward the end of pregnancy or in the first postpartal months in previously healthy women. Recent data suggest a central role of unbalanced peri-/postpartum oxidative stress that triggers the proteolytic cleavage of the nursing hormone prolactin (PRL) into a potent antiangiogenic, proapoptotic, and proinflammatory 16-kDa PRL fragment. This notion is supported by the observation that inhibition of PRL secretion by bromocriptine, a dopamine D2-receptor agonist, prevented the onset of disease in an animal model of PPCM and by first clinical experiences where bromocriptine seem to exert positive effects with respect to prevention or treatment of PPCM patients. Here, we highlight the current state of knowledge on diagnosis of PPCM, provide insights into the biology and pathophysiology of 16-kDa PRL and bromocriptine, and outline potential consequences for the clinical management and treatment options for PPCM patients. [less ▲]

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See detailThe 16-kilodalton N-terminal fragment of human prolactin is a potent inhibitor of angiogenesis
Clapp, Carmen; Martial, Joseph ULg; Guzman, Raphaël C et al

in Endocrinology (1993), 133(3), 1292-9

The formation of a new blood supply, angiogenesis, is an essential component of carcinogenesis and unrestricted tumor growth. A substance capable of inhibiting angiogenesis would be of considerable ... [more ▼]

The formation of a new blood supply, angiogenesis, is an essential component of carcinogenesis and unrestricted tumor growth. A substance capable of inhibiting angiogenesis would be of considerable therapeutic potential in the treatment of cancer. We previously reported that the 16-kilodalton N-terminal fragment of rat PRL (16K rPRL) was a potent inhibitor of capillary endothelial cell proliferation via a novel receptor. We now report that the nanomolar concentrations of recombinant human 16K PRL inhibit basal and basic fibroblast growth factor- or vascular endothelial growth factor-stimulated growth of bovine brain capillary endothelial cells. 16K human (h) PRL also inhibits stimulation of human umbilical vein endothelial cell proliferation by basic fibroblast growth factor. The organization of endothelial cells into capillary-like structures in type I collagen gels is also prevented by 16K hPRL. Furthermore, in an in vivo assay, the chick embryo chorioallantoic membrane assay, 16K hPRL as well as 16K rPRL were potent inhibitors of capillary formation. 16K hPRL, like 16K rPRL, maintains its biological activity as a partial PRL agonist at PRL receptors on mammary gland epithelial cells. These data demonstrate for the first time that the biological activity of 16K rPRL is not unique and that similar fragments of hPRL are active. The antiangiogenic activity of these molecules is conserved across avian and mammalian species. That 16K hPRL is a potent antiangiogenic factor in in vitro and an in vivo assay raises the exciting potential of this peptide being capable of inhibiting tumor growth. [less ▲]

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See detail1609, la révolution de l'observation
Nazé, Yaël ULg

Article for general public (2009)

Not Available

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See detail16K hPRL prevents angiogenesis by inducing both apoptosis and cell cycle arrest of endothelial cells
Tabruyn, Sébastien ULg; Sorlet, Catherine; Georges, Angélique et al

Poster (2003)

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