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See detailChronic kidney disease in Taiwan.
Delanaye, Pierre ULg; Cavalier, Etienne ULg; Krzesinski, Jean-Marie ULg

in Lancet (2008), 372(9654), 19501950-1

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See detailChronic low back pain. Good clinical practice (GCP)
Nielens, H.; Van Zundert, J.; Mairiaux, Philippe ULg et al

Report (2006)

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See detailChronic mastitis-affected cows display lower lipoxin levels than acute mastitis-affected cows
Boutet, Philippe ULg; Bureau, Fabrice ULg; Degand, Guy ULg et al

in Pflügers Archiv : European Journal of Physiology (2004), 447

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See detailChronic mastitis-affected cows display lower lipoxin levels than acute mastitis-affected cows
Boutet, Philippe ULg; Bureau, Fabrice ULg; Degand, Guy ULg et al

in Recueil "Le Médecin Vétérinaire du Québec" (2004), 34(1 et 2), 160

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See detailChronic measles virus infection of mouse nerve cells in vitro
Rentier, Bernard ULg; Claysmith, A. P.; Dubois-Dalcq, Monique et al

in Bishop, David H. L.; Compans, Richard W. (Eds.) The replication of negative strand viruses : proceedings of the 4th International Symposium on Negative Strand Viruses held October 26-November 1, 1980 at Frenchman’s Reef, Saint Thomas, U.S. Virgin Islands (1981)

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See detailChronic mucocutaneous herpes simplex virus and varicella zoster virus infections
Wauters, Odile ULg; Lebas, Eglantine ULg; Nikkels, Arjen ULg

in Journal of the American Academy of Dermatology (2012)

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See detailChronic non-cardiac cough in dogs
Clercx, Cécile ULg

in Proceedings of the 35th Annual World Small Animal Veterinary Association Congress (2010, June)

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See detailChronic obstructive pulmonary disease (COPD) can induce reversible cor pulmonale in horses
Amory, Hélène ULg; Christmann, U

in Journal of Equine Veterinary Science (1999), 19

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See detailChronic ozone exposure affects leaf senescence of adult beech trees: a chlorophyll fluorescence approach
Gielen, B.; Low, M.; Deckmyn, G. et al

in Journal of Experimental Botany (2007), 58(4), 785-795

Accelerated leaf senescence is one of the harmful effects of elevated tropospheric ozone concentrations ([O-3]) on plants. The number of studies dealing with mature forest trees is scarce however ... [more ▼]

Accelerated leaf senescence is one of the harmful effects of elevated tropospheric ozone concentrations ([O-3]) on plants. The number of studies dealing with mature forest trees is scarce however. Therefore, five 66-year-old beech trees (Fagus sylvatica L.) have been exposed to twice-ambient (2xambient) [O-3] levels by means of a free-air canopy O-3 exposure system. During the sixth year of exposure, the hypothesis of accelerated leaf senescence in 2xambient [O-3] compared with ambient [O-3] trees was tested for both sun and shade leaves. Chlorophyll (chl) fluorescence was used to assess the photosynthetic quantum yield, and chl fluorescence images were processed to compare functional leaf homogeneity and the proportion of O-3-injured leaf area (stipples) under ambient and 2xambient [O-3] regimes. Based on the analysis of chl fluorescence images, sun leaves of both ambient and 2xambient [O-3] trees had apparently developed typical necrotic O-3 stipples during high O-3 episodes in summer, while accelerated senescence was only observed with sun leaves of 2xambient [O-3] trees. This latter effect was indicated along with a faster decrease of photosynthetic quantum yield, but without evidence of changes in non-photochemical quenching. Overall, treatment effects were small and varied among trees. Therefore, compared with ambient [O-3], the consequence of the observed O-3-induced accelerated leaf senescence for the carbon budget is likely limited. [less ▲]

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See detailChronic rupture of abdominal aortic aneurysm manifesting as crural neuropathy.
Defraigne, Jean-Olivier ULg; SakalihasanN, Natzi ULg; LAVIGNE, Jean-Paul ULg et al

in Annals of Vascular Surgery (2001), 15(3), 405-11

Chronic rupture of abdominal aortic aneurysm (AAA) resulting in unusual clinical manifestations can occur if the resistance of structures surrounding the aorta is sufficient to contain hemorrhage. In this ... [more ▼]

Chronic rupture of abdominal aortic aneurysm (AAA) resulting in unusual clinical manifestations can occur if the resistance of structures surrounding the aorta is sufficient to contain hemorrhage. In this report, we describe five cases of chronic ruptured AAA in which the presenting feature was crural neuropathy. All patients were male with a mean age of 74 +/- 1.8 years. At the time of presentation, crural neuropathy had been ongoing for 3 to 9 weeks. In three cases, AAA was not initially suspected because an inadequate clinical examination was performed (not in the vascular surgery department) and because of the small diameter of the aorta in relation to the patient's morphology. Two patients had one episode of hypotension that was wrongly attributed to vagal attack. Abdominal CT scanning was always diagnostic of chronic rupture. In two cases, rupture was associated with erosion of the body of one or more vertebrae and laboratory evidence of inflammation, i.e., increase in sedimentation rate and fibrinogen level. The mean diameter of the AAA was 7.1 +/- 0.9 cm (range 5-10 cm). All patients underwent midline laparotomy, which was performed under emergency conditions in two cases, under semi-emergency conditions in one case, and electively in two cases. Perforation was consistently located on the posterolateral wall of the aorta and varied from 1 to 3 cm in length. Repair was performed using an aortobifemoral prosthesis in four cases, and a straight tube in one case. The patient who underwent emergency surgery died 4 days after the procedure. The remaining four patients recovered uneventfully and were discharged after 10 days. In the elderly, ruptured AAA should be included in the differential diagnosis of crural neuropathy. An episode of hypotension, regardless of its duration, in an elderly patient should be given serious consideration as a possible sign of ruptured AAA with ongoing retroperitoneal hemorrhage. [less ▲]

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See detailChronic septic arthritis of the carpus : Surgical approach
Touati, Kamal ULg; Sartelet, Arnaud ULg

Conference (2008, July 10)

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See detailChronic tension-type headache
Fumal, Arnaud ULg; Schoenen, Jean ULg

in Decker, B. C. (Ed.) Chronic daily headaches for clinicians (2005)

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See detailChronic tension-type headache: a multifactorial analysis suggesting disturbance of "limbic pathways" to the brain stem
Schoenen, Jean ULg; Bottin, D.; Juprelle, M. et al

in Clifford Rose, F. (Ed.) New Advances in Headache Research (1991)

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See detailChronic tension-type headache: what is new?
Fernandez-de-las-Penas, Cesar; Schoenen, Jean ULg

in Current Opinion in Neurology (2009), 22(3), 254-61

PURPOSE OF REVIEW: This review discusses current data on nosological boundaries related to diagnosis, pathophysiology and therapeutic strategies in chronic tension-type headache (CTTH). RECENT FINDINGS ... [more ▼]

PURPOSE OF REVIEW: This review discusses current data on nosological boundaries related to diagnosis, pathophysiology and therapeutic strategies in chronic tension-type headache (CTTH). RECENT FINDINGS: Diagnostic criteria of CTTH should be adapted to improve its sensitivity against migraine. It seems that mechanical pain sensitivity is a consequence and not a causative factor of CTTH. Recent evidence is modifying previous knowledge about relationships between muscle tissues and CTTH, suggesting a potential role of muscle trigger points in the genesis of pain. An updated pain model suggests that headache perception can be explained by referred pain from trigger points in the craniocervical muscles, mediated through the spinal cord and the trigeminal nucleus caudalis rather than only tenderness of the muscles themselves. Different therapeutic strategies, pharmacological, physical therapy, psychological and acupuncture, are generally used. The therapeutic efficacy of nonsteroidal anti-inflammatory drugs remains incomplete. The tricyclic antidepressants are the most used first-line therapeutic agents for CTTH. Surprisingly, few controlled studies have been performed and not all of them have found an efficacy superior to placebo. Further, there is insufficient evidence to support/refute the efficacy of physical therapy in CTTH. SUMMARY: Although there is an increasing scientific interest in CTTH, future studies incorporating subgroups of patients who will likely to benefit from a specific treatment (clinical prediction rules) should be conducted. [less ▲]

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See detailChronic tolerance to ethanol-induced sedation: Implication for age-related differences in locomotor sensitization
Quoilin, Caroline; Didone, Vincent ULg; Tirelli, Ezio ULg et al

in Alcohol (2013), 47(4), 317-322

The adolescent brain has been suggested to be particularly sensitive to ethanol-induced neuroadaptations, which in turn could increase the risk of youths for alcohol abuse and dependence. Sensitization to ... [more ▼]

The adolescent brain has been suggested to be particularly sensitive to ethanol-induced neuroadaptations, which in turn could increase the risk of youths for alcohol abuse and dependence. Sensitization to the locomotor stimulant effects of ethanol has often been used as an animal model of ethanol-induced neuroadaptations. Previously, we showed that young mice were more sensitive than adults to the locomotor sensitization induced by high ethanol doses. However, this effect could be due to age-related differences in chronic tolerance to the sedative effects of ethanol. The aim of the present study is to assess chronic tolerance to the sedative effects of ethanol in weaning 21-day-old (P21), adolescent 35-day-old (P35) and adult 63-day-old (P63) female Swiss mice. After a daily injection of saline or 4 g/kg ethanol during 6 consecutive days, all P21, P35 and P63 mice were injected with 4 g/kg ethanol and submitted to the loss of righting reflex procedure. Our results confirm that the sensitivity to the acute sedative effects of ethanol gradually increases with age. Although this schedule of ethanol injections induces significant age-related differences in ethanol sensitization, it did not reveal significant differences between P21, P35 and P63 mice in the development of a chronic ethanol tolerance to its sedative effects. The present results show that age-related differences in the development of ethanol sensitization cannot be explained by differences in chronic ethanol tolerance to its sedative effects. More broadly, they do not support the idea that ethanol-induced sensitization is a by-product of chronic ethanol tolerance. [less ▲]

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See detailChronic Toxic Hepatitis in Beef Calves due to Mycotoxins in Mixed Feed
Theron, Léonard ULg; Callebaut, Alfons; Bayrou, Calixte ULg et al

in Zemljic, Borut; Podpecan, Ozbalt; Zmljic-Jokhadar, Spela (Eds.) Proceedings of the XV middle European Buiatric Congress (2015, June 10)

Mycotoxins intoxination is an emerging disorder in Belgium, due to evolution of cultural practices and harvesting weather conditions. These intoxinations are difficult to diagnose for the vet practioners ... [more ▼]

Mycotoxins intoxination is an emerging disorder in Belgium, due to evolution of cultural practices and harvesting weather conditions. These intoxinations are difficult to diagnose for the vet practioners, since unspecific clinical signs, and their impact on ruminant disorder remains controversial. Although legal concentrations have been established for mycotoxins in the EU, farm forages are most of the time not tested (EU 2006/576/EC). In January 2015, a 500 calvings cross-bred Belgian blue cattle herd (BVDV free) referred a second (the first was directly sent to the rendering-plant) unexplained fatal case of jaundice on a 2 months-old calf to the Clinic for Ruminants of the University of Liège for necropsy. In 2013, a case of jaundice due to a Salmonella dublin hepatocholecystitis had previously diagnosed in this farm (Ronzoni et al., 2014), but so far preventions measures were implemented. Necropsy revealed generalized icterus, mild bilirubinuria, splenomegaly, but no precise etiology. On February a second 2 month old calf with jaundice is referred, lethargic, normothermic with a mild diarrhea, Calf shown generalized jaundice, anemia, elevation of leukocytes (neutrophils, eosinophils and monocytes), TGO, bilirubinemia, total biliary acids and Globulins (alpha2). He had also diminished erythrocytes and albuminemia. Copraemia, leptospirosis serology, leademia, hemoculture and pancreatic enzymes were within normal ranges, ruling out classic causes of jaundice in calf. Abdomen ultrasonography revealed a mild hyperechogenicity of the liver but no gall bladder modifications. Symptomatic treatment was based on symptoms, with fluidotherapy and choleretics. A third calf was referred three days after in a worst clinical condition, with also a severely modified liver enzymes, but no anemia. Three days after, this calf died and a necropsy revealed petechiae and hemorrhages in the abomasum, congestive mucosae in the distal bowel, white depot in the kidney medulla, modified urine and splenomegaly. Bile bacteriology and leptospirosis PCR was negative and liver histopathology revealed a severe histopathological liver degeneration associated with a disruption of the parenchyma and marked hyperplasia of the bile ducts compatible with chronic metabolic disorder. Meanwhile, the anemic calf recovered from anemia without any treatment after 4 days and was discharged from the Clinic for Ruminants. Considering the weird clinical patterns of these jaundice cases, and the fact that only calves from 2 to 3 months were affected, a nutritional origin as etiology was suspected. To test it, eight clinically healthy two to three months-old calves, of two different pens were sampled. TGO, GLDH, GGT or biliary acids were either modified and the values tended to increase with the age of the calves. The water was analyzed for classic toxics, and cultured for total germ content and everything were within recommended values. The calves fed with a milk replacer (30 % milk powder), and a commercial calf starter until one month of age. Then, they were given a mixed feed (containing cereal mix, cocoa, beet pulp, soja, maize), made at the farm from primary product/byproducts bought from different sources. The mix was tested for mycotoxins presence and ranged from 0.8 to 1.5 ppm of Desoxynivalenone (DON), and 115-215 ppb of Zearalenone (ZEA). The principal source of DON was maize (3.1 to 6.2 ppm), as for ZEA (0.3-0.6 ppm), but several compounds contained small dose of DON (0.2-0.7 ppm) for Barley feed, Tanned soja. Cacao contained 2.7 to 5.9 ppb of Ochratoxine (OTA). Nutritionnal recommendations were immediately given with a change in the source of maize and an addition of clay and yeast at 40g/calf/day and hay. Since, any other hypothetic origin to this progressive hepatic intoxination was demostrated, and that the doses founded, even if barely legal in the mix, are not accounted for toxic in the EU regulation, we believe that the calves were chronically exposed to these toxins. However, some mycotoxins experts still claim that various clinical signs would be observed in ruminants, if the rumen is partially defaunated, like in our case with the lake of forage. We propose that monitoring of subclinical liver health could be a key to screen DON effects. [less ▲]

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See detailChronic Toxicity Of Essential Oils Of 3 Local Aromatic Plants Towards Sitophilus Zeamais Motsch (Coleoptera : Curculionidae)
Ngamo, Tinkeu L.S.; Goudoum, A.; Ngassoum, Martin B. et al

in African Journal of Agricultural Research (2007), 2(4), 164-167

Detailed reference viewed: 35 (3 ULg)