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See detailCorrelation of phasic muscle strength and corticomotoneuron conduction time in multiple sclerosis.
van der Kamp, W.; MAERTENS DE NOORDHOUT, Alain ULg; Thompson, P. D. et al

in Annals of Neurology (1991), 29(1), 6-12

Central motor conduction times for the adductor pollicis muscle, the twitch force of that muscle to scalp magnetic motor cortex stimulation, and the maximum force of phasic voluntary contraction of the ... [more ▼]

Central motor conduction times for the adductor pollicis muscle, the twitch force of that muscle to scalp magnetic motor cortex stimulation, and the maximum force of phasic voluntary contraction of the same muscle were measured in 15 patients with multiple sclerosis. Two tests of manual dexterity of the same hand also were studied: the Purdue pegboard test, and the maximal frequency of a scissors movement of the thumb and index finger. The patients had normal strength or minimal weakness of the intrinsic muscles of the hand on clinical examination. The mean central motor conduction times for the adductor pollicis muscle for the patients were longer than normal, the peak twitch force of the adductor pollicis muscle evoked by cortical stimulation and the maximum force of a phasic voluntary contraction of the adductor pollicis muscle were smaller than normal. There were strong correlations between all these measures. Central motor conduction time in the patients was inversely correlated with voluntary phasic force and the twitch force after cortical stimulation. That is, the longer the central motor conduction time, the weaker the force. Prolonged central motor conduction time is likely to be accompanied by conduction block in corticomotoneuron pathways. The correlation of central motor conduction time with voluntary phasic force and the twitch force most likely reflects the degree of conduction block and temporal dispersion rather than delay in conduction per se. These results indicate that objective assessments of phasic muscle strength may reveal correlations with central motor conduction time that are not evident on conventional clinical examination which assesses tonic muscle contraction strength.(ABSTRACT TRUNCATED AT 250 WORDS) [less ▲]

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See detailA disorder of axonal development, necrotizing myopathy, cardiomyopathy and cataracts: A new familial disorder
Lyon, Gilles; Arita, Frenando; Le Galloudec, Eric et al

in Annals of Neurology (1990), 27

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See detailEffects of beta blockade on contingent negative variation in migraine.
MAERTENS DE NOORDHOUT, Alain ULg; Timsit-Berthier, M.; Timsit, M. et al

in Annals of Neurology (1987), 21(1), 111-112

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See detailContingent Negative Variation in Headache
MAERTENS DE NOORDHOUT, Alain ULg; Timsit-Berthier, Martine; Timsit, M. et al

in Annals of Neurology (1986), 19(1), 78-80

Contingent negative variation (CNV), an event-related slow cerebral potential, was analyzed in 79 consecutive headache patients. Compared to normal controls (n = 33), CNV did not differ in tension ... [more ▼]

Contingent negative variation (CNV), an event-related slow cerebral potential, was analyzed in 79 consecutive headache patients. Compared to normal controls (n = 33), CNV did not differ in tension headache (n = 21) or in combined headaches with a predominant tension component (n = 13). The mean amplitude of CNV was significantly (p less than 0.001) increased in migraine (n = 29) as well as in combined headache with predominant migraine (n = 16). All migraineurs were studied between attacks and without prophylactic treatment. CNV may be a useful diagnostic test in headache. Its increased amplitude in migraine might reflect central catecholaminergic hyperactivity. [less ▲]

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See detailThe effect of TRH on F waves recorded from antagonistic muscles in human subjects.
Delwaide, P. J.; Schoenen, Jean ULg

in Annals of Neurology (1985), 18(3), 366-7

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