References of "Wang, François-Charles"
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See detailLe syndrome du canal lent
Zeevaert, Bernard; WANG, François-Charles ULg; CRIELAARD, Jean-Michel ULg

Conference (2000)

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See detailNombre et taille des unités motrices dans la sclérose latérale amyotrophique
WANG, François-Charles ULg; DE PASQUA, Victor ULg; GERARD, Pascale ULg et al

in Magistris, Michel (Ed.) L’électroneuromyographie en l’an 2000 : mises au point (2000)

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See detailSingle motor unit H reflexes recorded in thenar muscles at rest.
Wang, François-Charles ULg; Delwaide, Paul ULg

in Muscle & nerve (1999), 22(2), 291-2

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See detailPosture et mouvement
Delwaide, Paul ULg; Maertens de Noordhout, Alain ULg; WANG, François-Charles ULg

in Held, Jean-Pierre; Dizien, Olivier (Eds.) Traité de Médecine Physique et de Réadaptation (1999)

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See detailNumber and relative size of thenar motor units in ALS patients: application of the adapted multiple point stimulation method.
Wang, François-Charles ULg; Delwaide, Paul ULg

in Electroencephalography and Clinical Neurophysiology (1998), 109(1), 36-43

In the present study, the adapted multiple point stimulation (AMPS) method was first applied to median innervated thenar muscles in 22 amyotrophic lateral sclerosis (ALS) patients who did not received any ... [more ▼]

In the present study, the adapted multiple point stimulation (AMPS) method was first applied to median innervated thenar muscles in 22 amyotrophic lateral sclerosis (ALS) patients who did not received any treatment. In all patients, a motor unit number estimate (MUNE) and an average surface-recorded motor unit action potential (S-MUAP) size have been derived even if the denervation was severe; and the results were reproducible. The thenar MUNE was less than the normal lower limit for age in 17 patients, and the mean MUNE (67.1 +/- 90.6) was significantly different from that estimated in control subjects (263.3 +/- 116.8). The mean S-MUAP size in the 22 ALS patients was 352.9 +/- 328.4 microV x ms versus 94.1 +/- 30.3 microV x ms in healthy volunteers. A control AMPS was achieved in 8 patients after 2 and 6 months of a glutamate-release antagonist (riluzole) treatment. The mean loss of motor units, based on control thenar MUNEs realized after 6 months of treatment, was 53%. In conclusion, we propose AMPS as a manageable, reproducible and non-invasive procedure which permits one to quantify peripheral denervation and to appreciate the effectiveness of collateral reinnervation in ALS patients. [less ▲]

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See detailElectrophysiological monitoring in clinical trials
Bril, Valérie; Ellison, R.; Ngo, Marguerite et al

in Muscle & Nerve (1998)

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See detailElectrophysiological classification of Guillain-Barré Syndrome: clinical associations and outcome
Hadden, R. D. M.; Cornblath, D. R.; Hughes, R. A. C. et al

in Annals of Neurology (1998)

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See detailRéflexe H unitaire
WANG, François-Charles ULg; CRIELAARD, Jean-Michel ULg

Conference (1998)

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Peer Reviewed
See detailLe syndrome du canal lent
Zeevaert, Bernard; WANG, François-Charles ULg; CRIELAARD, Jean-Michel ULg

Conference (1998)

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See detailMyasthenia gravis without chronic GVHD after allogeneic bone marrow transplantation.
Baron, Frédéric ULg; Sadzot, Bernard ULg; Wang, François-Charles ULg et al

in Bone Marrow Transplantation (1998), 22(2), 197-200

A 20-year-old man with aplastic anemia developed myasthenia gravis (MG) 7 months after bone marrow transplantation (BMT) from an HLA one locus-mismatched sister. Proximal muscle weakness (predominant in ... [more ▼]

A 20-year-old man with aplastic anemia developed myasthenia gravis (MG) 7 months after bone marrow transplantation (BMT) from an HLA one locus-mismatched sister. Proximal muscle weakness (predominant in the lower limbs) and dysphagia occurred without any other sign of graft-versus-host disease (GVHD), 1 month after cessation of immunosuppression with cyclosporine. The diagnosis of MG was based on clinical symptoms and on neurophysiologic investigations showing a significant increase of the Jitter in single-fiber electromyography and a significant decremental response during repetitive stimulation at slow rates, but antibodies against the acetylcholine receptor (AchRab) were negative. All clinical and neurophysiological signs normalized within 1 month of treatment with low-dose prednisolone and pyridostigmine, and the patient is perfectly well 1 year after cessation of all therapy. All cases of BMT-associated MG previously published are reviewed in comparison with ours. The originality of this new observation is that this case is the only one not associated with chronic GVHD and negative for AchRab. Alternatively, MG may have been the sole manifestation of chronic GVHD in this patient. [less ▲]

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