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See detailSyndromes canalaires rares du membre supérieur - Apport des bilans électrophysiologiques
Kaux, Jean-François ULg; MASSART, Nicolas ULg; TINANT, France ULg et al

in 32ème Séminaire de Traumatologie du Sport de l'AMDTS (2013, May)

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See detailL’examen électrophysiologique du nerf thoracique long (nerf de Charles Bell)
WANG, François-Charles ULg; Courtois, Anne-Catherine ULg; TINANT, France ULg

in Lettre du Neurologue (La) : le Courrier du Spécialiste (2009)

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See detailAn isokinetic eccentric programme for the management of chronic lateral epicondylar tendinopathy
Croisier, Jean-Louis ULg; Foidart-Dessalle, Marguerite ULg; Tinant, France ULg et al

in British Journal of Sports Medicine (2007), 41(4), 269-275

Background: Lateral epicondylitis represents a frequent overuse injury. In spite of many conservative treatment procedures, prolonged symptoms and relapse are frequently observed. Objective: To compare ... [more ▼]

Background: Lateral epicondylitis represents a frequent overuse injury. In spite of many conservative treatment procedures, prolonged symptoms and relapse are frequently observed. Objective: To compare the outcome of patients performing an isokinetic eccentric training with that of age-, gender-, activity-matched patients receiving a non-strengthening classical rehabilitation. Methods: Ninety-two patients with unilateral chronic lateral epicondylar tendinopathy (mean duration of symptoms 8 +/- 3 months) were assigned either to a control group (n = 46) or to an eccentrically trained group (n = 46). The control group underwent a passive standardised rehabilitation programme that excluded strengthening exercises. In addition to this programme, the trained group also performed eccentric exercises based on the repetitive lengthening of the active musculo-tendinous unit. The latter exercises started with submaximal contraction intensity and slow speed movement. Modalities were progressively intensified (increase in intensity contraction and speed movement) over a long priod of treatment. Programme effectiveness was assessed through pain score evaluation, a disability questionnaire, muscle strength measurement and ultrasonographic examination. Results: Compared to the non-strengthening control group, the following observations were made in the eccentrically trained group: (1) a significantly more marked reduction of pain intensity, mainly after one month of treatment; (2) an absence of strength deficit on the involved side through bilateral comparison for the forearm supinator and wrist extensor muscles; (3) an improvement of the tendon image as demonstrated by decreasing thickness and a recovered homogenous tendon structure; and (4) a more marked improvement in disability status during occupational, spare time and sports activities. Conclusion: These results highlight the relevance of implementing isokinetic adapted eccentric training in the management of chronic lateral epicondylar tendinopathy. [less ▲]

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See detailTravail excentrique sous-maximal sur dynamomètre isocinétique et traitement des épicondylites chroniques
Foidart-Dessalle, Marguerite ULg; Forthomme, Bénédicte ULg; Tinant, France ULg et al

in Actes du Congrès de la Société Royale Belge de Médecine Physique et de Réhabilitation (2002, November)

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See detailLa neuropathie avec hypersensibilite hereditaire a la pression ou neuropathie tomaculaire
Tinant, France ULg; Zeevaert, Bernard ULg; Benkirane, H. et al

in Revue Médicale de Liège (2002), 57(10), 651-4

Hereditary neuropathy liability to pressure palsies is characterized by recurring accesses of painless paralysis at the level of various nerves likely to be compressed. This affection remains ... [more ▼]

Hereditary neuropathy liability to pressure palsies is characterized by recurring accesses of painless paralysis at the level of various nerves likely to be compressed. This affection remains underdiagnosed because of its usually benign course, sometimes without any symptom. The diagnosis is supported by clinical and electrophysiological data associated with, in the majority of patients, a deletion of one of the alleles coding for protein PMP 22 on the level of the locus 17p11.2. [less ▲]

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See detailMultifocal motor and sensory neuropathy following whiplash injury
DIVE, Dominique ULg; NOEZ, Stéphanie ULg; ISERENTANT, Cynthia ULg et al

in Journal of Neurology (2001)

Detailed reference viewed: 32 (5 ULg)