References of "SCHUMACHER, Katharina"
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See detailEvaluation of the effectiveness of simulation of cardiac arrhythmias in children
Bragard, Isabelle ULg; Seghaye, Marie-Christine ULg; Baugnon, Thomas et al

Poster (2015, May)

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See detailEvaluation de l’efficacité de la simulation haute fidélité dans la prise en charge des troubles du rythme cardiaque chez l’enfant: étude pilote
SCHUMACHER, Katharina ULg; Seghaye, Marie-Christine ULg; Baugnon, Thomas et al

in Tijdschrift van de Belgische Kinderarts = Journal du Pédiatre Belge (2015, March), 17(1),

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See detailthe development care WEECARE reduces the stress in neonates
Battisti, Oreste ULg; NYAMUGABO MUNYERE NKANA, Kindja ULg; zigabe, serge et al

in Annales Africaine de Médecine (2015)

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See detailImplémentation d’un cours avec simulation haute fidélité pour la préparation des internes débutant leur formation en pédiatrie et en médecine d’urgence
SCHUMACHER, Katharina ULg; Seghaye, Marie-Christine ULg; Solowianiuk, Marie et al

in Tijdschrift van de Belgische Kinderarts = Journal du Pédiatre Belge (2015), 17(1),

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See detailMyocardial expression of cytokines influences postoperative outcome after cardiac surgery for congenital cardiac defects
Sprute, Johanna; Heying, Ruth; Buding, Brigitte et al

Conference (2014, January)

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See detailCardiac malformations in neonates born from mothers with gestational diabetes
Antole, Nathalie; Jacquemart, Caroline; GKIOUGKI, Evangelia ULg et al

Conference (2014, January)

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See detailMyocardial cardiotrophin-1 is differentially induced in congenital cardiac defects depending on hypoxemia.
Heying, Ruth; Qing, Ma; SCHUMACHER, Katharina ULg et al

in Future cardiology (2014), 10(1), 53-62

Aim: Cardiotrophin-1 (CT-1) is upregulated by hypoxemia and hemodynamic overload and is characterized by potent hypertrophic and protective properties on cardiac cells. This study aimed to investigate ... [more ▼]

Aim: Cardiotrophin-1 (CT-1) is upregulated by hypoxemia and hemodynamic overload and is characterized by potent hypertrophic and protective properties on cardiac cells. This study aimed to investigate whether CT-1 is differentially induced in the myocardium of infants with congenital cardiac defects depending on hypoxemia. Methods & results: Infants with Tetralogy of Fallot (n = 8) or with large nonrestrictive ventricular septal defect (n = 8) undergoing corrective surgery were investigated. Expression of CT-1 was assessed at mRNA and protein levels in the right atrial and ventricular myocardium. The activation of the STAT-3 and VEGF were measured. Degradation of cardiac troponin-I served as a marker of myocardial damage. CT-1 was detected in all patients with levels negatively correlating to the arterial oxygen saturation. Higher CT-1 expression in Tetralogy of Fallot patients was associated with activation of the JAK/STAT pathway and higher cardiac troponin-I degradation. Conclusion: CT-1 may mediate myocardial hypertrophy and dysfunction in infants with congenital cardiac defects, particularly in those with hypoxemia. [less ▲]

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See detailInterleukin-6 as a marker of inflammatory related post-operative myocardial dysfunction
von der Stück, H.; SCHUMACHER, Katharina ULg; Buding, B. et al

Conference (2013, May)

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