References of "Meyer, G"
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See detailBovine herpesvirus 5 infection
Meyer, G.; Thiry, Julien ULg; Thiry, Etienne ULg

in Lefèvre, P. C.; Blancou, J.; Chermette, R. (Eds.) et al Infectious and Parasitic Diseases of Livestock (2010)

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See detailIBR and BVD control : the key to successful herd management
Makoschey, B.; Franken, P.; Mars, J. M. H. et al

in Berliner und Munchener Tierarztliche Wochenschrift (2010), 123

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See detailEmbryo transfer as a tool for experimental reproduction of ovine herds.
Rizzo, H.; François, D.; Fassier, T. et al

in Ciência Animal Brasileira (2009), Suppl 1

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See detailBluetongue control in Europe - New challenges and achievements
Makoschey, B.; MacLachlan, J.; Van Wuijckhuise, L. et al

in Berliner und Munchener Tierarztliche Wochenschrift (2009), 122

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See detailVaccination of calves using the BRSV nucleocapsid protein in a DNA prime-protein boost strategy stimulates cell-mediated immunity and protects the lungs against BRSV replication and pathology.
Letellier, Carine; Boxus, Mathieu ULg; Rosar, Laurent et al

in Vaccine (2008), 26(37), 4840-8

Respiratory syncytial virus (RSV) is a major cause of respiratory disease in both cattle and young children. Despite the development of vaccines against bovine (B)RSV, incomplete protection and ... [more ▼]

Respiratory syncytial virus (RSV) is a major cause of respiratory disease in both cattle and young children. Despite the development of vaccines against bovine (B)RSV, incomplete protection and exacerbation of subsequent RSV disease have occurred. In order to circumvent these problems, calves were vaccinated with the nucleocapsid protein, known to be a major target of CD8(+) T cells in cattle. This was performed according to a DNA prime-protein boost strategy. The results showed that DNA vaccination primed a specific T-cell-mediated response, as indicated by both a lymphoproliferative response and IFN-gamma production. These responses were enhanced after protein boost. After challenge, mock-vaccinated calves displayed gross pneumonic lesions and viral replication in the lungs. In contrast, calves vaccinated by successive administrations of plasmid DNA and protein exhibited protection against the development of pneumonic lesions and the viral replication in the BAL fluids and the lungs. The protection correlated to the cell-mediated immunity and not to the antibody response. [less ▲]

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See detailConcepts in the prevention of bovine respiratory disease.
Makoschey, B.; Lekeux, Pierre ULg; Lacroux, C. et al

in Berliner und Munchener Tierarztliche Wochenschrift (2008), 121(11-12), 446-449

The bovine respiratory disease (BRD) complex requires further research both, to fully understand the disease from the different perspectives as well as to develop new tools and strategies for vaccination ... [more ▼]

The bovine respiratory disease (BRD) complex requires further research both, to fully understand the disease from the different perspectives as well as to develop new tools and strategies for vaccination and treatment was the conclusion at a recent BRD symposium in Rome, Italy. A group of scientist across Europe followed the invitation of Prof. E. Thiry (University of Liège, Belgium) to convene for a 2 days workshop type symposium sponsored by Intervet/Schering-Plough Animal Health (Fig. 1 – group picture) [less ▲]

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See detailBovine Herpesvirus Type 1 Glycoprotein H Is Essential for Penetration and Propagation in Cell Culture
Meyer, G.; Hanon, E.; Georlette, D. et al

in Journal of General Virology (The) (1998), 79((Pt 8)), 1983-7

Bovine herpesvirus type 1 (BHV-1) glycoprotein H (gH) is a structural component of the virion which forms a complex with glycoprotein gL. To study the role of BHV-1 gH in the virus infectious cycle, a gH ... [more ▼]

Bovine herpesvirus type 1 (BHV-1) glycoprotein H (gH) is a structural component of the virion which forms a complex with glycoprotein gL. To study the role of BHV-1 gH in the virus infectious cycle, a gH null mutant was constructed in which the gH coding sequences were deleted and replaced by the Escherichia coli lacZ cassette. The BHV-1 gH null mutant was propagated in trans-complementing MDBK cells, stably transfected with plasmid pMEP4 containing the BHV-1 gH gene under the control of the inducible mouse metallothionein promoter. Experiments with the BHV-1 gH null mutant showed that gH is essential in the infectious cycle of the virus and is specifically involved in virus entry and cell-to-cell spread. The lack of infectivity of virions devoid of gH is not due to a defect in attachment. Moreover, PEG-induced fusion of virions to target cells provides evidence that BHV-1 gH is required for virion penetration. [less ▲]

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