References of "Chellappa, Sarah Laxhmi"
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See detailCircadian Rhythm Sleep Disorder: Genetic and Environmental Factors
Chellappa, Sarah Laxhmi ULg; Viola, Antoine; Mongrain, Valerie

in Kushida, Clete (Ed.) Encyclopaedia of Sleep (in press)

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See detailCircadian and homeostatic regulation of sleepiness and cognition and its neuronal underpinnings
Schmidt, Christina; Chellappa, Sarah Laxhmi ULg; Cajochen, Christian

in Garbarino, Sergio (Ed.) Sleepiness and Human Impact Assessment (in press)

Detailed reference viewed: 17 (2 ULg)
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See detailTwo time pieces for sleep regulation: the circadian clock and the homeostatic hourglass
Cajochen, Christian; Schmidt, Christina; Chellappa, Sarah Laxhmi ULg

in Garbarino, Sergio (Ed.) Sleepiness and Human Impact Assessment (in press)

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See detailEvolution of Treatment and Investigative Approaches in Sleep Medicine
Ly, Julien; Chellappa, Sarah Laxhmi ULg; MAQUET, Pierre ULg

in Billiard, Michael (Ed.) Sleep Medicine: a comprehensive guide (in press)

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See detailNeurophysiological basis of sleep and wakefulness
Chellappa, Sarah Laxhmi ULg; Schmidt, Christina; Cajochen, Christian

in Garbarino, Sergio (Ed.) Sleepiness and Human Impact Assessment (in press)

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See detailHuman cortical excitability depends on time spent awake and circadian phase
Ly, Julien ULg; Gaggioni, Giulia ULg; Chellappa, Sarah Laxhmi ULg et al

Scientific conference (2014, October 04)

At any point in time, human performance results from the interaction of two main factors: a circadian signal varying with the time of the day and the sleep need accrued throughout the preceding waking ... [more ▼]

At any point in time, human performance results from the interaction of two main factors: a circadian signal varying with the time of the day and the sleep need accrued throughout the preceding waking period. But what’s happen at the cortical cerebral level? We used a novel technique coupling transcranial magnetic stimulation with electroencephalography (TMS/EEG) to assess the influence of time spent awake and circadian phasis on human cortical excitability. Twenty-two healthy young men underwent 8 TMS/EEG sessions during a 28 hour sleep deprivation protocole. We found that cortical excitability depends on both time spent awake and circadian phasis. [less ▲]

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See detailAutomatic artifact detection for whole-night polysomnographic sleep recordings
Coppieters't Wallant, Dorothée ULg; Chellappa, Sarah Laxhmi ULg; Gaggioni, Giulia ULg et al

Poster (2014, September 17)

Detecting of bad channels and artifacts for whole-night polysomnographic recordings is very time consuming and tedious. We therefore developed an automatic procedure to automatize this job.

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See detailHuman cortical excitability depends on time awake and circadian phase
Ly, Julien ULg; Chellappa, Sarah Laxhmi ULg; Gaggioni, Giulia ULg et al

Conference (2014, September 17)

At any point in time, human performance results from the interaction of two main factors: a circadian signal varying with the time of the day and the sleep need accrued throughout the preceding waking ... [more ▼]

At any point in time, human performance results from the interaction of two main factors: a circadian signal varying with the time of the day and the sleep need accrued throughout the preceding waking period. But what’s happen at the cortical cerebral level? We used a novel technique coupling transcranial magnetic stimulation with electroencephalography (TMS/EEG) to assess the influence of time spent awake and circadian phasis on human cortical excitability. Twenty-two healthy young men underwent 8 TMS/EEG sessions during a 28 hour sleep deprivation protocole. We found that cortical excitability depends on both time spent awake and circadian phasis. [less ▲]

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See detailHuman cortical excitability depends on time spent awake and circadian phase
Ly, Julien ULg; Chellappa, Sarah Laxhmi ULg; Gaggioni, Giulia ULg et al

Conference (2014, September 17)

At any point in time, human performance results from the interaction of two main factors: a circadian signal varying with the time of the day and the sleep need accrued throughout the preceding waking ... [more ▼]

At any point in time, human performance results from the interaction of two main factors: a circadian signal varying with the time of the day and the sleep need accrued throughout the preceding waking period. But what’s happen at the cortical cerebral level? We used a novel technique coupling transcranial magnetic stimulation with electroencephalography (TMS/EEG) to assess the influence of time spent awake and circadian phasis on human cortical excitability. Twenty-two healthy young men underwent 8 TMS/EEG sessions during a 28 hour sleep deprivation protocole. We found that cortical excitability depends on both time spent awake and circadian phasis. [less ▲]

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See detailAutomatic biorythms description from actigraphic data
González y Viagas, Miguel ULg; Ly, Julien ULg; Gaggioni, Giulia ULg et al

Poster (2014, September)

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See detailCortical excitability dynamics of during sleep deprivation set PVT performance
Borsu, Chloé; Gaggioni, Giulia ULg; Ly, Julien ULg et al

Poster (2014, September)

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See detailPrior light history impacts on higher order cognitive brain function
Chellappa, Sarah Laxhmi ULg; Ly, Julien; Meyer, Christelle ULg et al

Conference (2014, June 17)

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See detailThe circadian system sets the temporal organization of basic human neuronal function
Chellappa, Sarah Laxhmi ULg; Ly, Julien; Gaggioni, Giulia et al

Conference (2014, June 16)

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See detailDynamics of human cortical ensembles are set by circadian system and sleep homeostasis
Chellappa, Sarah Laxhmi ULg; Ly, Julien; Gaggioni, Giulia et al

Conference (2014)

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See detailCortical excitability dynamics of during sleep deprivation set PVT performance
Borsu, Chloé; Gaggioni, Giulia ULg; Ly, Julien et al

Conference (2014)

Detailed reference viewed: 12 (4 ULg)
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See detailLight modulation of human sleep depends on a polymorphism in the clock gene PER3
Chellappa, Sarah Laxhmi ULg; Viola, Antoine; Schmidt, Christina ULg et al

in Behavioural Brain Research (2014), 271

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See detailLight modulation of human sleep depends on a polymorphism in the clock gene Period3.
Chellappa, Sarah Laxhmi ULg; Viola, Antoine U.; Schmidt, Christina ULg et al

in Behavioural brain research (2014), 271

Non-image-forming (NIF) responses to light powerfully modulate human physiology. However, it remains scarcely understood how NIF responses to light modulate human sleep and its EEG hallmarks, and if there ... [more ▼]

Non-image-forming (NIF) responses to light powerfully modulate human physiology. However, it remains scarcely understood how NIF responses to light modulate human sleep and its EEG hallmarks, and if there are differences across individuals. Here we investigated NIF responses to light on sleep in individuals genotyped for the PERIOD3 (PER3) variable-number tandem-repeat (VNTR) polymorphism. Eighteen healthy young men (20-28 years; mean+/-SEM: 25.9+/-1.2) homozygous for the PER3 polymorphism were matched by age, body-mass index, and ethnicity. The study protocol comprised a balanced cross-over design during the winter, during which participants were exposed to either light of 40lx at 6500K (blue-enriched) or light at 2500K (non-blue enriched), during 2h in the evening. Compared to light at 2500K, light at 6500K induced a significant increase in all-night NREM sleep slow-wave activity (SWA: 1.0-4.5Hz) in the occipital cortex for PER3(5/5) individuals, but not for PER3(4/4) volunteers. Dynamics of SWA across sleep cycles revealed increased occipital NREM sleep SWA for virtuallyall sleep episode only for PER3(5/5) individuals. Furthermore, they experienced light at 6500K as significantly brighter. Intriguingly, this subjective perception of brightness significantly predicted their increased occipital SWA throughout the sleep episode. Our data indicate that humans homozygous for the PER3(5/5) allele are more sensitive to NIF light effects, as indexed by specific changes in sleep EEG activity. Ultimately, individual differences in NIF light responses on sleep may depend on a clock gene polymorphism involved in sleep-wake regulation. [less ▲]

Detailed reference viewed: 12 (0 ULg)