References of "Sandersen, Charlotte"
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See detailEmergence of bovine Ehrlichiosis in Belgium
Guyot, Hugues ULg; Vandeputte, Sébastien ULg; Sandersen, Charlotte ULg et al

Conference (2005)

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See detailClues for Differential Diagnosis of atypical myopathy
Votion, Dominique ULg; Amory, Hélène ULg; Sandersen, Charlotte ULg et al

in Proceeding of the Maastricht International Congress on Equine Medicine (MICEM) (2004, December)

Atypical myopathy (AM), also called “atypical myoglobinuria”, is a frequently and rapidly fatal myopathy of unknown origin occurring sporadically in grazing horses. As opposed to the exertional ... [more ▼]

Atypical myopathy (AM), also called “atypical myoglobinuria”, is a frequently and rapidly fatal myopathy of unknown origin occurring sporadically in grazing horses. As opposed to the exertional rhabdomyolysis syndrome (ERS), clinical signs of AM are not induced by exercise. The condition has been reported in several European countries including Belgium, France, Germany and Great Britain. Clinical signs of AM are characterised by muscular weakness, stiffness, recumbency, sweating and when urine is observed, myoglobinuria. These signs are characteristic but not pathognomonic of the disease; the differential diagnosis of sudden weakness, severe myopathy and/or unexpected death includes several neurogenic and myopathic disorders. The main pathologies that share clinical similarities with AM include the acute form of grass sickness (GS), acute piroplasmosis, botulism, ERS, the hyperkalemic periodic paresis (HPP), nutritional myopathy (NM; i.e. vitamin E and/or selenium deficiency), plants or drugs (i.e. ionophores intoxication) intoxication, tetanus and postanaesthesia myopathy. This report aims at reviewing key facts in history, clinical signs, clinical examination and laboratory findings that contribute towards the diagnosis of AM and/or invalidate the diagnosis of other pathologies. [less ▲]

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See detailDoppler Echocardiographic Reference Values in Healthy Donkeys
Amory, Hélène ULg; Bertrand, P.; Delvaux, Véronique ULg et al

in Matthews, N. S.; Taylor, T. S. (Eds.) Veterinary Care of Donkeys (2004)

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See detailCase report : A suspicion of cortico-cerebral necrosis in a Belgian Blue herd after ingestion of moulded silage
Guyot, Hugues ULg; Sandersen, Charlotte ULg; Aliaoui, Hamani et al

Poster (2004)

After ingestion of moulded beet pulp silage, cases of cortico-cerebral necrosis (CCN) and mortalities have been observed in a Belgian Blue (BB) herd. Contamination with Paecilomyces spp., a mould that ... [more ▼]

After ingestion of moulded beet pulp silage, cases of cortico-cerebral necrosis (CCN) and mortalities have been observed in a Belgian Blue (BB) herd. Contamination with Paecilomyces spp., a mould that produces byssochlamic acid, malformins and patulin, has been proven. Among these toxins, patulin is known to have cancerogenic, immunosuppressive and tremorgenic effects, but also acts on the respiratory and digestive systems. Twenty-five days after progressive introduction of beet pulp silage into the ration of a dual purpose BB herd, most of the animals showed diminished appetite, salivation and decreased milk production. All 35 cows were reluctant to consume the beet pulp silage, but continued to eat grass silage voluntarily. Seven of them showed anorexia and nervous symptoms, like head pressing and blindness. Four animals died within 1 week after onset of neurological symptoms. No necropsy has been performed, since legislation does not allow post-mortem examination of the central nervous system in the field. The three survivors had been treated successfully with thiamine (10 mg/kg, IV, TID) and recovered completely within five days. After the beet pulp silage had been identified as causative agent, it had been removed from the animals’ ration and no more clinical case has been observed. Four weeks later, the same beet pulp silage has been reintroduced into the animals’ ration and provoked again diminished appetite, salivation and a decrease in milk production in most of the animals. Clinical signs were also suggesting lead poisoning but any contact with lead containing material could have been excluded. Silage was obviously moulded and analysis revealed the presence of 1.6 million CFU Paecilomyces spp./g of silage. Although no further investigation has been made to identify the mycotoxins, an intoxication with patulin has been suspected, since other mycotoxins produced by these species are less toxic. Although it has not been described that CCN can be induced by ingestion of Paecilomyces spp., it seems that there is a close relation between ingestion of Paecilomyces-contaminated silage and observed clinical signs in this herd. [less ▲]

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See detailDoppler echocardiographic repercussions of a 12 week treadmill training period in Standardbreds
Amory, Hélène ULg; De Moffarts, Brieuc; Art, Tatiana ULg et al

in 43rd Congress of the British Equine Veterinary Association (2004)

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See detailBiochemical findings associated with atypical myopathy in grazing horses
Votion, Dominique ULg; Delguste, Catherine ULg; Amory, Hélène ULg et al

in Pflügers Archiv : European Journal of Physiology (2004), 447

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See detailDuodenitis/Proximal Jejunitis- a retrospective study of 27 cases
Lopez, D; Delguste, Catherine ULg; Sandersen, Charlotte ULg et al

Poster (2004)

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See detailRetrospective study of 41 cases of mitral insufficiency in horses
Bertrand, D; Maurin, E; Sandersen, Charlotte ULg et al

Poster (2004)

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See detailA case of Vitamin K3 induced renal toxicosis in a horse
Sandersen, Charlotte ULg; Cassart, Dominique ULg; Bertrand, P et al

in Tierärztliche Praxis Kleintiere (2004)

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See detailCerebrospinal fluid level of protein S100 beta in healthy horse : a preliminary study
Lopez, D; De Moffarts, Brieuc; Delguste, Catherine ULg et al

in Tierärztliche Praxis Kleintiere (2004)

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See detailTraining-induced changes in aerobic capacity assessed by VO2max and echocardiography in standardbred horses
Art, Tatiana ULg; De Moffarts, Brieuc; Sandersen, Charlotte ULg et al

in Lindsey, A. Abeyasekere; Frances, J.Barr (Eds.) Handbook of the 43rd Congress of the British Equine Veterinary Association (2004)

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See detailEvaluation of plasma carboxy-terminal cross-linking telopeptide of type I collagen concentration in horses.
Carstanjen, Bianca; Hoyle, Nicholas R; Gabriel, Annick ULg et al

in American Journal of Veterinary Research (2004), 65(1), 104-9

OBJECTIVE: To evaluate a human assay for quantification of carboxy-terminal cross-linking telopeptide of type I collagen (CTX-I), assess the influence of age on plasma CTX-I concentration, investigate the ... [more ▼]

OBJECTIVE: To evaluate a human assay for quantification of carboxy-terminal cross-linking telopeptide of type I collagen (CTX-I), assess the influence of age on plasma CTX-I concentration, investigate the relationship between plasma CTX-I and serum osteocalcin concentrations, and determine whether concentrations of plasma CTX-I or serum osteocalcin fluctuate in circadian manner in horses. HORSES: 75 clinically normal horses. PROCEDURE: Cross-reactivity between equine serum CTX-I and CTX-I antibodies in an automated electrochemiluminescent sandwich antibody assay (ECLIA) was evaluated via a specificity test (ie, dilution test) and recovery calculation. Serum osteocalcin concentration was measured with an equine-specific osteocalcin radioimmunoassay. To analyze diurnal variations in plasma CTX-I and serum osteocalcin concentrations, blood samples were obtained hourly during a 24-hour period. RESULTS: Results of the dilution test indicated good correlation (r > 0.99) between expected serum CTX-I concentrations and measured serum CTX-I concentrations. The calculated CTX-I recovery was 97.6% to 109.9%. Plasma CTX-I and serum osteocalcin concentrations were correlated. Plasma CTX-I concentration was inversely correlated with age of the horse. No significant circadian variations in plasma CTX-I and serum osteocalcin concentrations were detected. CONCLUSIONS AND CLINICAL RELEVANCE: Results suggest that the fully automated CTX-I ECLIA can be used for evaluation of plasma and serum samples from horses and may be a useful tool to monitor bone metabolism changes. Horses in this study did not have notable diurnal fluctuations in serum osteocalcin and plasma CTX-I concentrations. [less ▲]

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See detailAtypical Myopathy (Atypical Myoglobinuria)
Votion, Dominique ULg; Amory, Hélène ULg; Demoulin, Vincent ULg et al

in IVIS Reviews in Veterinary Medicine (2004)

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See detailLa myopathie atypique des équidés: particularités cliniques, examens complémentaires et hypothèses étiologiques.
Votion, Dominique ULg; Delguste, Catherine ULg; Amory, Hélène ULg et al

in Journée AVEF (Association des Vétérinaires Equins Français) (2003, October)

La myopathie atypique (MA) des équidés, encore appelée myoglobinurie atypique des chevaux au pré, est un syndrome caractérisé par l’apparition soudaine de faiblesse musculaire, de raideur, de tremblements ... [more ▼]

La myopathie atypique (MA) des équidés, encore appelée myoglobinurie atypique des chevaux au pré, est un syndrome caractérisé par l’apparition soudaine de faiblesse musculaire, de raideur, de tremblements et de sudation profuse généralisée ou localisée. L’animal adopte rapidement une position en décubitus sternal ou latéral. Lorsque de l’urine est émise, celle-ci a une couleur brune « chocolatée ». L’examen clinique révèle fréquemment de la tachycardie, de l’hypothermie et de la dyspnée. Le dosage de l’activité sérique de la créatinine kinase contribue à la présomption de MA en démontrant une destruction musculaire massive mais le diagnostic définitif repose nécessairement sur l’examen histologique des muscles intervenant dans la posture et la respiration, muscles généralement atteints lors de cette maladie ainsi que sur l’examen du muscle cardiaque où des lésions de dégénérescence sont occasionnellement observées. Diverses hypothèses étiologiques sont en cours d’investigation et les plus probables incriminent l’action d’une mycotoxine ou d’une toxine d’origine bactérienne, ingérée ou produite dans le tractus digestif. Néanmoins, une carence nutritionnelle n’est pas exclue. Quelle que soit la cause, il semble que des conditions climatiques défavorables favorisent le déclenchement des symptômes. La récolte de données épidémiologiques permettra de mieux définir les moyens de prévention de cette maladie souvent fatale. [less ▲]

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See detailStress echocardiography in horses: comparison of cardiac output during incremental dobutamine infusion in atropinised and non-atropinised ponies
Sandersen, Charlotte ULg; Detilleux, Johann ULg; Delguste, Catherine ULg et al

in Proceedings of the 42d Annual Congress of the British Equine Veterinairy Association (2003, September)

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See detailClinicopathological data in equids suffering from ante-hepatic, hepatic and post-hepatic diseases
Amory, Hélène ULg; Perron, MF; Sandersen, Charlotte ULg et al

in Proceedings of the 42th Annual Congress of the British Equine Veterinary Association (BEVA) (2003)

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See detailPrognostic value of clinical signs and blood parameters in equids suffering from acute hepatic insufficiency: a retrospective study on 31 cases
Amory, Hélène ULg; Perron, MF; Sandersen, Charlotte ULg et al

in Proceedings of the 21st ACVIM annual meeting, Charlotte, North Carolina (2003)

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