References of "Rentier, Bernard"
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See detailEffect of DNA photosensitization mediated by promazine derivatives on transcription in vitro
Decuyper, J.; Piette, Jacques ULg; Rentier, Bernard ULg et al

in Archives Internationales de Physiologie, de Biochimie et de Biophysique (1984), 92(2), 18

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See detailNatural infection of Swiss mice by the Mouse Mammary Tumor Virus (MMTV). 2. Studies on the pathway of infection
Hainaut, P.; Vaira, Dolorès ULg; Calberg-Bacq, Claire Michelle et al

in Archives Internationales de Physiologie, de Biochimie et de Biophysique (1984), 92(1), 28

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See detailModèles expérimentaux pour l'étude des infections virales du système nerveux
Rentier, Bernard ULg

in Bulletin et Mémoires de l'Académie Royale de Médecine de Belgique (1984), 139(3), 218-226

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See detailMyelination and myelinating cells in culture
Moonen, Gustave ULg; Rentier, Bernard ULg

in Bulletin de la Société Belge d'Ophtalmologie (1983), 208(PT 1), 57-62

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See detailExperimental models to study measles virus persistence in the nervous system
Rammohan, K. W.; Dubois-Dalcq, M.; Rentier, Bernard ULg et al

in Progress in Neuropathology (1983), 5

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See detailFibronectin promotes rat Schwann cell growth and motility
Baron-Van Evercooren, Anne; Kleinman, Hinda K.; Seppa, H. E. et al

in Journal of Cell Biology (1982), 93(1), 211-216

Techniques are now available for culturing well characterized and purified Schwann cells. Therefore, we investigated the role of fibronectin in the adhesion, growth, and migration of cultured rat Schwann ... [more ▼]

Techniques are now available for culturing well characterized and purified Schwann cells. Therefore, we investigated the role of fibronectin in the adhesion, growth, and migration of cultured rat Schwann cells. Double-immunolabeling shows that, in primary cultures of rat sciatic nerve, Schwann cells (90%) rarely express fibronectin, whereas fibroblasts (10%) exhibit a granular cytoplasmic and fibrillar surface-associated fibronectin. Secondary cultures of purified Schwann cells do not express fibronectin. Exogenous fibronectin has a small effect on promoting the adhesion of Schwann cells to the substrate and does not significantly affect cell morphology, but it produced a surface fibrillar network on fibronectin on the secondary Schwann cells. Tritiated thymidine autoradiography revealed that addition of fibronectin to the medium, even at low concentrations, markedly stimulates Schwann cell proliferation, in both primary and secondary cultures. In addition, when cell migration was measured in a Boyden chamber assay, fibronectin was found to moderately, but clearly, stimulate directed migration or chemotaxis. [less ▲]

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See detailAcute and persistent viral infections of differentiated nerve cells
Dubois-Dalcq, Monique; Rentier, Bernard ULg; Hooghe-Peters, Elisabeth L. et al

in Reviews of Infectious Diseases (1982), 4(5), 999-1014

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See detailSchwann cells in vitro
Rentier, Bernard ULg

in Archives de Biologie (1982), 93

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See detailEtude de l'infection rougeoleuse in vitro et de sa persistance dans les cellules nerveuses en culture
Rentier, Bernard ULg

Thèse d’agrégation de l’enseignement supérieur (1982)

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See detailStructural studies on acute and persistent viral infections of nerve cells
Dubois-Dalcq, Monique; Rentier, Bernard ULg; Hooghe-Peeters, E. et al

in Fifth international congress of virology : abstracts = 5e Congrès international de virologie : abstracts, Strasbourg, France, August 2-7, 1981 (1981)

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See detailChronic measles virus infection of mouse nerve cells in vitro
Rentier, Bernard ULg; Claysmith, A. P.; Dubois-Dalcq, Monique et al

in Bishop, David H. L.; Compans, Richard W. (Eds.) The replication of negative strand viruses : proceedings of the 4th International Symposium on Negative Strand Viruses held October 26-November 1, 1980 at Frenchman’s Reef, Saint Thomas, U.S. Virgin Islands (1981)

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See detailEtude de l'infection rougeoleuse in vitro et de sa persistance dans les cellules nerveuses en culture
Rentier, Bernard ULg

in Bulletin de l'Institut Pasteur (1981), 79(2), 107-170

Measles virus can infect neurons in culture. In mouse fetal neurons, infection is spontaneously persistent. Reactivation occurs very rarely. In human fetal neurons, infection is persistent if cells are ... [more ▼]

Measles virus can infect neurons in culture. In mouse fetal neurons, infection is spontaneously persistent. Reactivation occurs very rarely. In human fetal neurons, infection is persistent if cells are cultured in human serum. However, if serum-free medium is used, infection becomes lytic and productive.Latency in mouse neurons is apparently due to the species barrier while latency in human neurons is induced by anti-measles virus antibodies contained in the serum. This might explain the difference between measles encephalitis and subacute sclerosing panencepalitis (SSPE), with presence of anti-measles antibody in the brain in the latter and not in the former. [less ▲]

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See detailStructure and behavior of rat primary and secondary Schwann cells in vitro
Dubois-Dalcq, Monique; Rentier, Bernard ULg; Baron-Vanevercooren, Anne et al

in Experimental Cell Research (1981), 131(2), 283-297

The structure and motility of isolated rat primary (I) Schwann cells (SC) have been compared to that of subcultured (II) SC during and after mitotic stimulation. I SC contain myelin components which ... [more ▼]

The structure and motility of isolated rat primary (I) Schwann cells (SC) have been compared to that of subcultured (II) SC during and after mitotic stimulation. I SC contain myelin components which persist for 2 weeks in serum-free medium while they rapidly disappear in medium containing serum and high glucose concentration. These components were never detected in II SC. Both I SC and II SC after their mitotic phase are spindle-shaped, contain many intermediate and actin filaments, have no basement membrane but show intense migratory and undulatory activities. Rare fibroblasts in I cultures are recognized by their extremely variable shape, the presence of Thy 1.1 antigen in their membrane and their intense edge ruffling alternating with abrupt translocation. In contrast, I SC movements consist of intracellular translocation of nuclei along SC processes, which retract and extend constantly, and in slow rhythmic undulation episodes (2.3 ± 0.2/min) alternating with migration at 135 ± 50 μ/h. The total number of these episodes per day in serum-free medium is rigorously identical for different cells (166.3 ± 0.2) and this uniformity of frequency suggests a genotypic basis. Cycles, consisting of an undulation episode followed by a resting interval, have mean durations of 8.6 ± 4.1 min and a sharp peak of occurrence at 6 min, with exponential distribution of the longer periods. Motility of II SC is considerably inhibited during mitotic stimulation by cholera toxin and a pituitary extract while SC phenotype has changed to a flat multipolar cell with prominent Golgi and ribosomes. Migration is reduced to 24 ± 2 μ/h and only 2% of the SC show pulsations of the same periodicity as the I SC undulations. A dramatic increase in pulsation frequency occurs 6–12 h after removal of mitogenic factors when 80% of II SC start pulsating twice as fast for 2–3 days. When mitoses cease, SC quickly recover their SC phenotype with rhythmic undulations while migration speed increased to 92 ± 20 μ/h. Thus, in spite of dramatic modification of shape, structure and behavior during mitotic stimulation, SC subsequently recover their unique motility pattern which might be essential for their myelinating function [less ▲]

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See detailBehavior and structure of rat primary and secondary Schwann cells in vitro
Dubois-Dalcq, Monique; Rentier, Bernard ULg; Baron, A. et al

in Journal of Neuropathology and Experimental Neurology (1980), 39(3), 350

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See detailPreservation of pulsory and migratory activities of rat Schwann cells before and after mitotic stimulation
Rentier, Bernard ULg; Dubois-Dalq, Monique; Baron-Van Evercooren, A.

in Abstracts - Society for Neuroscience (1980)

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See detailSelective and persistent measles virus infection of mouse neurons in vitro
Rentier, Bernard ULg; Claysmith, A. P.; Dubois-Dalcq, Monique et al

in Journal of Neuropathology and Experimental Neurology (1980), 39(3), 185-185

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See detailScanning and transmission electron microscopy study of antibody-dependent lymphocyte-mediated cytotoxicity on measles virus-infected cells
Rentier, Bernard ULg; Wallen, William C.

in Infection and Immunity (1980), 30(1), 303-315

The structural events related to antibody-dependent lymphocyte-mediated cytotoxicity (ADLC) have been studied on measles virus-infected cells using human peripheral blood lymphocytes (PBL) and anti ... [more ▼]

The structural events related to antibody-dependent lymphocyte-mediated cytotoxicity (ADLC) have been studied on measles virus-infected cells using human peripheral blood lymphocytes (PBL) and anti-measles virus serum. The first event in ADLC was a recognition process occurring within 15 min after contact between the infected cells and lymphocytes. Plasma membrane and microvilli of adsorbed PBL were specifically attached to virus-induced ridges over nucleocapsids and to viral buds. After 30 min, a fraction of adsorbed PBL (K cells) changed shape and extended long filipodia toward the target cells which, in turn, showed long villi contacting the PBL. At 4 h, when cytotoxicity as measured by chromium release was maximum, K cells had flattened and numerous blebs and ruffles formed on their surface. The K-cell alterations varied in intensity with the type of measles-infected target cell, but frequently the K cells appeared irreversibly damaged. T- and non-T-cell fractions were separated, and in situ erythrocyte rosettes were used as markers for subpopulations which were easily recognized by scanning electron microscopy. Most of the cytotoxic K cells were identified as non-T cells carrying Fc receptors for immunoglobulin G. However, a small subpopulation of cells bearing both sheep erythrocyte and Fc receptors was also found to be involved in ADLC by chromium release assay as well as by electron microscopy. Some of these interacting T cells extended a long uropod on the target cell, but their intracellular structure remained unaltered through ADLC, in contrast with the other T cells and the non-T killer cells. This suggests that perhaps some T killer cells might remain functional after the cytotoxic interaction with a target cell. [less ▲]

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