References of "Krzesinski, Jean-Marie"
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See detailPharma clinics. Comment je traite ... un trouble de la kaliémie
Neven, Ingrid; Krzesinski, Jean-Marie ULg

in Revue Médicale de Liège (2000), 55(1), 4-7

The treatment of dyskalemia must be primarily etiological and later symptomatic. When moderate but significant hypokalemia exists (K < 3 mmol/l or 3.5 mmol/l with cardiac disease), oral K supplements must ... [more ▼]

The treatment of dyskalemia must be primarily etiological and later symptomatic. When moderate but significant hypokalemia exists (K < 3 mmol/l or 3.5 mmol/l with cardiac disease), oral K supplements must be given. The intravenous route must be reserved for emergency or impossible oral administration. Acute and severe hyperkalemia (K > 7 mmol/l) must be first corrected by different intravenous measures with secondary oral intervention. In chronic and/or moderate hyperkalemia (K > 5.5 mmol/l), the oral route for treatment is prefered. In any case, the research of the cause (mainly drug induced) is fundamental. [less ▲]

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See detailIron monitoring under vitamin C therapy in hemodialysed patients treated with erythropoietin
Bovy, Christophe ULg; Dubois, Bernard ULg; Weekers, Laurent ULg et al

in Nephrology Dialysis Transplantation (2000), 15(9), 156

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See detailMesure de la pression artérielle en dehors du cabinet médical: intérêts et limites
Krzesinski, Jean-Marie ULg; Saint-Remy, Annie ULg

in Revue Médecine Générale (2000), 176

Standardized blood pressure self measurement with approved devices allows the patient's involvement in the diagnosis and provides information on various parameters.

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See detailLes sartans face aux inhibiteurs de l'enzyme de conversion de l'angiotensine en présence d'une hypertension artérielle
Krzesinski, Jean-Marie ULg; Sternon, J.

in Revue Médicale de Bruxelles (2000), 21(3), 170-177

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See detailEvaluation of the wrist cuff oscillometric blood pressure measurement in clinical practice
Saint-Remy, Annie ULg; Moonen, Martial; Krzesinski, Jean-Marie ULg et al

in Journal of Hypertension (Supplement) (2000), 18(suppl 2), 55

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See detailPharma clinics. Le médicament du mois. La lercanidipine (Zanidip)
Krzesinski, Jean-Marie ULg

in Revue Médicale de Liège (1999), 54(10), 832-834

Lercanidipine (Zanidip) is a new calcium antagonist belonging to the dihydropyridine family suitable for the first-line treatment of hypertension. This molecule displays a high specificity and selectivity ... [more ▼]

Lercanidipine (Zanidip) is a new calcium antagonist belonging to the dihydropyridine family suitable for the first-line treatment of hypertension. This molecule displays a high specificity and selectivity for vascular smooth muscle cells and has, in spite of a short plasma half-life, a long duration of action due to its liposolubility. The usual and once a day dose to treat high blood pressure of any grade is 10 mg, which if needed could be increased to 20 mg once a day. It has a gradual onset of effect on blood pressure, thus leading to a good tolerability and hopefully a better compliance in the treatment of hypertension. No adaptation of the dose is needed in older patients or in patients with moderate renal or liver impairment. [less ▲]

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See detailHypertension et grossesse
Krzesinski, Jean-Marie ULg

in Revue Médicale de Liège (1999), 54(5), 415-423

High blood pressure during pregnancy (BP > or = 140/90 mmHg) is sometimes already noted before conception, with usually a good prognosis (although it could predispose to preeclampsia). alpha-methyldopa is ... [more ▼]

High blood pressure during pregnancy (BP > or = 140/90 mmHg) is sometimes already noted before conception, with usually a good prognosis (although it could predispose to preeclampsia). alpha-methyldopa is the best treatment when needed (agents blocking the renin angiotensin system are not recommended). Preeclampsia, a form of hypertension noted after 20 weeks of gestation with proteinuria is a more serious condition (BP > or = 140/90 mmHg or increase in BP from the 1st trimester > or = 25/15 mmHg). It is generated by placental ischemia and creates maternal endothelial lesions which in turn decrease the blood flow to placenta leading to maternal and fetal syndromes. Hospitalisation is mandatory. No measure other than delivery is known to attenuate or reverse its progression. Treating hypertension during pregnancy (when blood pressure > or = 170/110 mmHg) aims at preventing maternal risk (stroke or eclampsia) but has few effect on foetal lesions. Prevention of this syndrome, which represents the first secondary cause of hypertension, is until now disappointing. [less ▲]

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See detailComment j'explore ... une suspicion de sténose d'artère rénale
Krzesinski, Jean-Marie ULg; Turatzinze, Léopold

in Revue Médicale de Liège (1999), 54(8), 702-704

Renal artery stenosis can present 2 clinical pictures, sometimes associated and potentially treatable, due mainly to atherosclerosis, but also to fibromuscular dysplasia. The first possible presentation ... [more ▼]

Renal artery stenosis can present 2 clinical pictures, sometimes associated and potentially treatable, due mainly to atherosclerosis, but also to fibromuscular dysplasia. The first possible presentation is renovascular hypertension which represents 1% of unselected hypertensive populations. The second possible presentation is ischemic renal disease, which represent 10% of the new indications for dialysis therapy and has a 50% mortality rate at 3 years. To explore this disease, arteriography, the gold standard, must be reserved for confirmation and treatment. Hypertension induced by the stenosis is best approached by captopril renal MAG 3 scintigraphy. Measurement of the stenosis severity can be performed either by duplex ultrasonography, magnetic resonance angiography or spinal CT angiography according to patients'characteristics, local experience and facilities. This disease must be searched in the presence of renal insufficiency of unknown cause or refractory hypertension. [less ▲]

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See detailComment j'explore ... les troubles de la calcémie
Janssens, Lise; Krzesinski, Jean-Marie ULg

in Revue Médicale de Liège (1999), 54(9), 764-769

Mechanisms for calcium homeostasis are complex but their understanding is important before investigating the calcium disorders. Hypercalcaemia is often due to hyperparathyroidism or cancers. The ... [more ▼]

Mechanisms for calcium homeostasis are complex but their understanding is important before investigating the calcium disorders. Hypercalcaemia is often due to hyperparathyroidism or cancers. The diagnostis must thus be rapid and a treatment decided in a global approach. Hypocalcaemia, more frequently noted, is often due to a general disorder with hypoalbuminemia or chronic renal failure. [less ▲]

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See detailComment j'explore ... un trouble de la kaliémie
Neven, Ingrid; Krzesinski, Jean-Marie ULg

in Revue Médicale de Liège (1999), 54(12), 943-947

Potassium is the most important intracellular cation,playing a role in neuromuscular excitability. Dyskaliema is common. The consequence of this ionic perturbation could be serious related to its ... [more ▼]

Potassium is the most important intracellular cation,playing a role in neuromuscular excitability. Dyskaliema is common. The consequence of this ionic perturbation could be serious related to its magnitude. Thus a quick etiological approach and a medical intervention are needed to rapidly correct this potentially lethal ionic disturbance and prevent its recurrence. [less ▲]

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See detailPharma clinics. Comment je traite ... les troubles de la calcémie
Janssens, Lise; Krzesinski, Jean-Marie ULg

in Revue Médicale de Liège (1999), 54(10), 782-785

Treatment of both hypercalcemia and hypocalcemia depends on the underlying disorder, the magnitude of the calcium abnormalities and the severity of symptoms. In the case of hypercalcemia, there is a broad ... [more ▼]

Treatment of both hypercalcemia and hypocalcemia depends on the underlying disorder, the magnitude of the calcium abnormalities and the severity of symptoms. In the case of hypercalcemia, there is a broad selection of effective medications, especially the biphosphonates. Treatment of hypocalcemia relies on the calcium intake and often vitamin D supplementation. For both abnormalities, the underlying cause must be treated, if possible, to definitively restore normocalcemia. [less ▲]

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See detailPharma clinics. Comment je traite ... la sténose d'artère rénale
Krzesinski, Jean-Marie ULg; Turatzinze, Léopold

in Revue Médicale de Liège (1999), 54(9), 719-721

Renal artery stenosis is mainly due to atherosclerosis, but also to fibromuscular dysplasia. Treatment can consist eisther of angioplasty (± stent) or of surgical revascularization. Hypertension induced ... [more ▼]

Renal artery stenosis is mainly due to atherosclerosis, but also to fibromuscular dysplasia. Treatment can consist eisther of angioplasty (± stent) or of surgical revascularization. Hypertension induced by atherosclerotic disease is rarely cured, but more easily controlled. The renal function is often improved and thus this disease must be searched in the presence of renal insufficiency of unknown cause or refractory hypertension. [less ▲]

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See detailActualisation de la prise en charge de l'hypertension artérielle
Krzesinski, Jean-Marie ULg

in Revue Médicale de Liège (1999), (54), 683-687

The International Society of Hypertension and the World Heath Organisation have recently published new guidelines for the management of hypertension. Epidemiologic studies have shown that management of ... [more ▼]

The International Society of Hypertension and the World Heath Organisation have recently published new guidelines for the management of hypertension. Epidemiologic studies have shown that management of hypertension still remains insatisfactory. The aim of the treatment is to reduce the cardiovascular morbidity and mortality by normalizing blood pressure. The target blood pressure under treatment must be less than 140/90 mmHg, and even lower for diabetic or chronic renal failure patients. In parallel, all cardio-vascular risk factors have to be corrected to decrease the global cardio-vascular risk. [less ▲]

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See detailLes diuretiques
Krzesinski, Jean-Marie ULg

in I.N.A.M.I consensus (1999)

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See detailLes antagonistes des récepteurs de l'angiotensine II
Krzesinski, Jean-Marie ULg

in Vaisseaux, Coeur, Poumons (1998), 3(4), 116-119

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See detailSystème rénine-angiotensine II-aldostérone: quoi de neuf en 1998?
Krzesinski, Jean-Marie ULg

in Médisphère (1998), 91(12-13),

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See detailRelationships between left ventricular hypertrophy and the amplitude of the dipping
Saint-Remy, Annie ULg; moonen, martial; Krzesinski, Jean-Marie ULg et al

Conference (1997, October 04)

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See detailIsolated systolic and diastolic dippers, an intermediate subgroup between dippers and non-dippers
Moonen, martial; Saint-Remy, Annie ULg; Krzesinski, Jean-Marie ULg et al

Conference (1997, October 04)

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See detailLa néphropathie épidémique a Hantavirus
Weekers, Laurent ULg; Biessaux, Yves ULg; Lamproye, Anne ULg et al

in Revue Médicale de Liège (1997), 52(8), 515-519

L'Entre-Sambre-et-Meuse a été le siège d'une épidémie de néphropathie épidémique à Hantavirus (NE) en 199293. Cinq cas ont été rencontrés au CHU de Liège en moins d'un an. Le virus responsable de cette ... [more ▼]

L'Entre-Sambre-et-Meuse a été le siège d'une épidémie de néphropathie épidémique à Hantavirus (NE) en 199293. Cinq cas ont été rencontrés au CHU de Liège en moins d'un an. Le virus responsable de cette affection appartient à la famille dès Hantavirus dont il existe huit sérotypes aux caractéristiques propres en terme de vecteur (rongeurs), de distribution géographique et de pathogénicité. Les pathologies induites chez l'homme vont de la néphropathie épidémique à Hantavirus - d'évolution le plus souvent bénigne- au syndrome pulmonaire à Hantavirus (SPH) - fréquemment mortel - en passant par la fièvre hémorragique avec syndrome rénal (FHSR) de sévérité intermédiaire. Dans nos contrées, on rencontre le sérotype Puumala dont le vecteur est le campagnol roussâtre. L'homme s'infecte par inhalation de particules contaminées et développe dans un pourcentage non précisé des cas, après une période d'incubation de 1 à 3 semaines, un tableau clinique de néphropathie épidémique à Hantavirus. Celui-ci se caractérise par l'apparition brutale d'une fièvre, de myalgies diffuses, de douleurs abdominales et/ou lombaires et de céphalées. Apparaissent ensuite à des degrés divers: nausées et vomissements, oligurie, myopie aiguë, toux,diathèse hémorragique, diarrhée... Biologiquement, on observe l'association d'une insuffisance rénale aiguë et d'une thrombopénie. L'évolution est spontanément favorable dans les deux à trois semaines. Le diagnostic suggéré par la clinique sera confirmé par la sérologie. La physiopathologie de l'insuffisance rénale et de la thrombopénie fait intervenir des interactions complexes entre l'hôte et le virus. Il n'existe pas de thérapeutique spécifique pour la néphropathie épidémique à Hantavirus. Les formes plus sévères (SPH et FHSR) peuvent bénéficierd'un traitement par Ribavirineen iv. [less ▲]

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