References of "Delanaye, Pierre"
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See detailVariation of PTH and bone biomarkers in hemodialysis patients.
DELANAYE, Pierre ULg; Warling, X; Moonen, M et al

Poster (2015)

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See detailVitamine D et maladie rénale chronique
DELANAYE, Pierre ULg; CAVALIER, Etienne ULg

in Benhamou, CL; Souberbielle, JC (Eds.) La vitamine D chez l’adulte (2015)

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See detailGuidelines about CKD classification: a provocative point of view
DELANAYE, Pierre ULg

Conference (2015)

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See detailEvaluation de la fonction rénale
DELANAYE, Pierre ULg

Conference given outside the academic context (2015)

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See detailPlace de la vitamine D native dans le traitement des patients dialysés
DELANAYE, Pierre ULg

Conference given outside the academic context (2015)

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See detailPourquoi et comment corriger les anomalies de la PTH ?
DELANAYE, Pierre ULg

Conference given outside the academic context (2015)

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See detailRituximab (MabThera®) : nouveau médicament dans les vascularites associées à ANCA
VON FRENCKELL, Christian ULg; DELANAYE, Pierre ULg

in Revue Médicale de Liège (2015), 70(2), 92-100

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See detailExploration de la fonction glomérulaire rénale : estimation du débit de filtration glomérulaire
Maillard, N; DELANAYE, Pierre ULg; Mariat, C

in Néphrologie & Thérapeutique (2015), 11(1), 54-67

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See detailAn Age-Calibrated Classification of Chronic Kidney Disease.
Glassock, RJ; DELANAYE, Pierre ULg; El-Nahas, M

in JAMA : Journal of the American Medical Association (2015), 314(6), 559-560

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See detailSafety of living kidney donation: another brick in the wall…and a solid (physiologic) one.
DELANAYE, Pierre ULg; Mariat, C; Glassock, RJ

in American Journal of Kidney Diseases (2015), 66(1), 1-3

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See detailThe myth of the future burden of CKD in United States.
DELANAYE, Pierre ULg; El-Nahas, M; Glassock, RJ

in American Journal of Kidney Diseases (2015), 66(1), 171-172

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See detailLifetime Risk of CKD: What Does It Really Mean?
DELANAYE, Pierre ULg; Glassock, RJ

in Clinical Journal of the American Society of Nephrology (2015), 10(9), 1504-1506

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See detailGlomerular Filtration Rate and Aging: Another Longitudinal Study--A Long Time Coming!
DELANAYE, Pierre ULg; Glassock, rj

in Nephron (2015), 131(1), 1-4

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See detailAbnormal glomerular filtration rate in children, adolescents and young adults starts below 75 mL/min/1.73 m2
Pottel, Hans; Hoste, Liesbeth; DELANAYE, Pierre ULg

in Pediatric Nephrology : Journal of the International Pediatric Nephrology Association (2015), 30(5), 821-828

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See detailKDIGO Guidelines and Kidney Transplantation: Is the cystatin-C Based Recommendation relevant?
Masson, I; Maillard, N; CAVALIER, Etienne ULg et al

in American Journal of Transplantation (2015), 15(8), 2211-4

The KDIGO guidelines propose a new approach to diagnose chronic kidney disease (CKD) based on estimated glomerular ®ltration rate (GFR). In patients with a GFR value comprised between 45 and 59 mL/ min/1 ... [more ▼]

The KDIGO guidelines propose a new approach to diagnose chronic kidney disease (CKD) based on estimated glomerular ®ltration rate (GFR). In patients with a GFR value comprised between 45 and 59 mL/ min/1.73m2 as estimated by the CKD-EPI creatinine equation (eGFRcreat), it is suggested to con®rm the diagnosis with a second estimation using the CKD-EPI cystatin C-based equations (eGFRcys/eGFRcreat-cys). We sought to determine whether this new diagnostic strategy might extend to kidney transplant recipients (KTR) and help to identify those with decreased GFR. In 670 KTR for whom a measured GFR was available, we simulated the detection of CKD using the two-steps approach recommended by the guidelines in comparison to the conventional approach relying on creatinine equation. One hundred forty-®ve patients with no albuminuria had eGFRcreat between 45 and 59 mL/ min/1.73m2. Among them, 23% had inulin clearance over 60 mL/min/1.73m2 and were thus incorrectly classi®ed as CKD patients. When applying the Kidney Disease: Improving Global Outcomes (KDIGO) strategy, 138 patients were con®rmed as having a GFR below 60 mL/min with eGFRcreat-cys. However, 21% of them were misclassi®ed in reference to measured GFR. Our data do no not support the use of cystatin C as a con®rmatory test of stage 3A CKD in KTR. [less ▲]

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See detailSystematic Analysis of two cystatin C assays using samples of 2057 older adults - The Berlin initiative study
Ebert, natalie; DELANAYE, Pierre ULg; Martus, Peter et al

Poster (2014, November)

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See detailImpact of the dialysis membrane on the Vitamin D metabolims markers
CAVALIER, Etienne ULg; DUBOIS, Bernard ULg; Urena, Pablo et al

Poster (2014, November)

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See detailCalibration and precision of serum creatinine and plasma cystatin C measurement: impact on the estimation of glomerular filtration rate
DELANAYE, Pierre ULg; CAVALIER, Etienne ULg; Cristol, Jean-Paul et al

in Journal of Nephrology (2014), 27(5), 467-75

Serum creatinine (SCr) is the main variable for estimating glomerular filtration rate (GFR). Due to interassay differences, the prevalence of chronic kidney disease (CKD) varies according to the assay ... [more ▼]

Serum creatinine (SCr) is the main variable for estimating glomerular filtration rate (GFR). Due to interassay differences, the prevalence of chronic kidney disease (CKD) varies according to the assay used, and calibration standardization is necessary. For SCr, isotope dilution mass spectrometry (IDMS) is the gold standard. Systematic differences are observed between Jaffe and enzymatic methods. Manufacturers subtract 0.30 mg/dl from Jaffe results to match enzymatic results (‘compensated Jaffe method’). The analytical performance of enzymatic methods is superior to that of Jaffe methods. In the original Modification of Diet in Renal Disease (MDRD) equation, SCr was measured by a Jaffe Beckman assay, which was later recalibrated. A limitation of this equation was an underestimation of GFR in the high range. The Chronic Kidney Disease Epidemiology (CKD-EPI) consortium proposed an equation using calibrated and IDMS traceable SCr. The gain in performance was due to improving the bias whereas the precision was comparable. The CKD-EPI equation performs better at high GFR levels (GFR[60 ml/ min/1.73 m2). Analytical limitations have led to the recommendation to give a grade ([60 ml/min/1.73 m2) rather than an absolute value with the MDRD equation. By using both enzymatic and calibrated methods, this cutoff-grade could be increased to 90 ml/min/1.73 m2 (with MDRD) and 120 ml/min/1.73 m2 (with CKD-EPI). The superiority of the CKD-EPI equation over MDRD is analytical, but the precision gain is limited. IDMS traceable enzymatic methods have been used in the development of the Lund– Malmo¨ (in CKD populations) and Berlin Initiative Study equations (in the elderly). The analytical errors for cystatin C are grossly comparable to issues found with SCr. Standardization is available since 2011. A reference method for cystatin C is still lacking. Equations based on standardized cystatin C or cystatin C and creatinine have been proposed. The better performance of these equations (especially the combined CKD-EPI equation) has been demonstrated. [less ▲]

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