References of "Charlier, Corinne"
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See detailDrogues stupéfiantes et sécurité routière en Belgique
Charlier, Corinne ULg; Verstraete, A.; Maes, V. et al

in Toxicorama (1998), X

Detailed reference viewed: 12 (0 ULg)
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See detailToxiques et analyses biologiques en toxicologie hospitalière
Capolaghi, B.; Charlier, Corinne ULg; Feuillu, A. et al

in Toxicorama (1998), X

Detailed reference viewed: 11 (1 ULg)
Peer Reviewed
See detailLe contrôle thérapeutique dans le traitement de substitution à la méthadone
Charlier, Corinne ULg

in Annales de Toxicologie Analytique (1998)

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See detailInfluence des résidus de pesticides sur la Santé de l'Homme
Charlier, Corinne ULg

in Acta Clinica Belgica (1998)

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See detailPsychotropes, alcool et sécurité routière en Belgique
Charlier, Corinne ULg

Conference (1998)

Detailed reference viewed: 11 (3 ULg)
See detailDiscrepancy in hemoglobin A1c results obtained by HPLC and CE: influence of carbamyl HB
Gougnard, Th; Chapelle, Jean-Paul ULg; Charlier, Corinne ULg et al

Poster (1997, October 18)

Detailed reference viewed: 14 (0 ULg)
Peer Reviewed
See detailLe monitoring thérapeutique des antidépresseurs
Charlier, Corinne ULg; Ansseau, Marc ULg; Gougnard, Thierry et al

in Revue Médicale de Liège (1997), 52(5), 336-44

Detailed reference viewed: 45 (8 ULg)
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See detailLes pesticides: effets endocriniens et cancérogènes chez l'Homme
Charlier, Corinne ULg; Plomteux, Guy ULg

in Toxicorama (1997), IX

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See detailManifestations indésirables des antidépresseurs
Charlier, Corinne ULg; Ansseau, Marc ULg; Pinto, Emmanuel ULg et al

in Toxicorama (1997), (Spécial LYON), 310-314

Detailed reference viewed: 10 (3 ULg)
See detailLa ciclosporine A
Charlier, Corinne ULg

Conference (1997)

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See detailDrogues et Sécurité Routière en Belgique
Charlier, Corinne ULg

Conference (1997)

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See detailAlcool et drogues au volant
Charlier, Corinne ULg

Conference (1997)

Detailed reference viewed: 13 (1 ULg)
See detailQuantification of drugs in plasma of injured drivers
Charlier, Corinne ULg

Conference (1997)

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See detailAltération de l'état de vigilance, alcool et médicaments
Charlier, Corinne ULg

Conference (1997)

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See detailTherapeutic monitoring of antidepressant drugs
Charlier, Corinne ULg

Conference (1997)

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See detailAccidents immunoallergiques dus aux médicaments
Charlier, Corinne ULg

Conference (1997)

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See detailLe monitoring des antidépresseurs
Charlier, Corinne ULg

Conference (1997)

Detailed reference viewed: 14 (2 ULg)
Full Text
Peer Reviewed
See detailEnhancement of Tamoxifen-Induced E-Cadherin Function by Ca2+ Channel Antagonists in Human Breast Cancer Mcf7/6 Cells
Charlier, Corinne ULg; Bruyneel, E.; Lechanteur, Chantal ULg et al

in European Journal of Pharmacology (1996), 317(2-3), 413-6

Despite its intensive use in adjuvant breast cancer therapy for more than 30 years, the exact mechanisms of action of tamoxifen have not yet been fully characterized. Tamoxifen was recently shown to ... [more ▼]

Despite its intensive use in adjuvant breast cancer therapy for more than 30 years, the exact mechanisms of action of tamoxifen have not yet been fully characterized. Tamoxifen was recently shown to restore the E-cadherin function of human breast cancer MCF7/6 cells and to suppress their invasive phenotype. Because tamoxifen interacts with targets implicated in Ca2+ homeostasis, we explored the possibility that the restoration of E-cadherin function in MCF7/6 cells induced by this drug could be affected by Ca2+ modulators. Two different Ca2+ channel antagonists (verapamil and nifedipine) potentiated the effect of tamoxifen on E-cadherin function, as evaluated with a fast cell aggregation assay. These molecules decreased the tamoxifen concentration needed to restore the E-cadherin function from 10(-6) M to 10(-7) M. When incubated with a Ca2+ channel agonist, Bay K8644 (methyl-1,4-dihydro-2,6-dimethyl-3-nitro-4-(2-trifluoro-methylphenyl)- pyridine-5-carboxylate), the effect of tamoxifen on E-cadherin function was completely abolished. These results demonstrate that the restoration of the E-cadherin function induced by tamoxifen depends, at least in part, on a Ca2+ pathway, and support the evidence of an effect of tamoxifen on Ca(2+)-dependent mechanisms. Our data also suggest that Ca2+ channel modulators could make it possible to decrease the dose of tamoxifen administered to patients without reducing the therapeutic effects. [less ▲]

Detailed reference viewed: 32 (7 ULg)
See detailNew trends in clinical toxicology
Charlier, Corinne ULg; Plomteux, Guy ULg

Conference (1996, October)

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